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Ryan C. Fink

Researcher at St. Cloud State University

Publications -  35
Citations -  1958

Ryan C. Fink is an academic researcher from St. Cloud State University. The author has contributed to research in topics: Escherichia coli & Gene. The author has an hindex of 21, co-authored 33 publications receiving 1616 citations. Previous affiliations of Ryan C. Fink include Max Planck Society & North Carolina State University.

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BugBase predicts organism-level microbiome phenotypes

TL;DR: BugBase is presented, an algorithm that predicts organism-level coverage of functional pathways as well as biologically interpretable phenotypes such as oxygen tolerance, Gram staining and pathogenic potential, within complex microbiomes using either whole-genome shotgun or marker gene sequencing data.
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Molecular evolution and structure--function relationships of the superoxide dismutase gene families in angiosperms and their relationship to other eukaryotic and prokaryotic superoxide dismutases.

TL;DR: The findings suggest that the 5' region of these genes played a pivotal role in the evolution of function of these enzymes, and it is critical to understand the physiological differences between the SODs in response to different stresses in order to compare their functions and evolutionary history.
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Elderberry flavonoids bind to and prevent H1N1 infection in vitro

TL;DR: The Direct Binding Assay established that flavonoids from the elderberry extract bind to H1N1 virions and, when bound, block the ability of the viruses to infect host cells.
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FNR Is a Global Regulator of Virulence and Anaerobic Metabolism in Salmonella enterica Serovar Typhimurium (ATCC 14028s)

TL;DR: The role of FNR as a positive regulator of motility, flagellar biosynthesis, and pathogenesis was confirmed by showing that the isogenic fnr mutant is nonmotile, lacks flagella, is attenuated in mice, and does not survive inside macrophages.
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Transcriptional responses of Escherichia coli K-12 and O157: H7 associated with lettuce leaves

TL;DR: The data suggested that the interaction of E. coli K-12 and O157:H7 with undamaged lettuce leaves likely is initiated via attachment to the leaf surface using curli fibers, a downward shift in their metabolism, and the suppression of biofilm formation.