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Salvatore Torrisi

Researcher at National Institutes of Health

Publications -  28
Citations -  1348

Salvatore Torrisi is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Stria terminalis & Prefrontal cortex. The author has an hindex of 17, co-authored 26 publications receiving 1121 citations. Previous affiliations of Salvatore Torrisi include Semel Institute for Neuroscience and Human Behavior & University of California, Los Angeles.

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Frontal-amygdala connectivity alterations during emotion downregulation in bipolar I disorder.

TL;DR: Evidence is provided that dysfunction in the neural networks responsible for emotion regulation, including the prefrontal cortex, cingulate, and subcortical structures, are present in BPI subjects, even while euthymic.
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Differences in resting corticolimbic functional connectivity in bipolar I euthymia.

TL;DR: Resting state functional connectivity in the brain between key emotion regulation regions in bipolar I disorder is examined to delineate differences in coupling from healthy subjects.
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fMRI Functional Connectivity Applied to Adolescent Neurodevelopment

TL;DR: This review describes task-based and resting-state functional magnetic resonance imaging (fMRI) analytic strategies, but it focuses on iFC findings from resting- state data to describe general developmental trajectories of brain network organization.
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Controlling automatic imitative tendencies: interactions between mirror neuron and cognitive control systems.

TL;DR: Effective connectivity analysis exploring the interactions between these regions, suggests a role for the mPFC and ACC in imitative conflict detection and the anterior insula in conflict resolution processes, which may occur through interactions with the frontal node of the human mirror neuron system (MNS).
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Regional fMRI Hypoactivation and Altered Functional Connectivity During Emotion Processing in Nonmedicated Depressed Patients With Bipolar II Disorder

TL;DR: It is suggested that bipolar II depression is characterized by reduced regional orbitofrontal and limbic activation and altered connectivity in a fronto-temporal circuit implicated in working memory and emotional learning.