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Moriah E. Thomason

Researcher at New York University

Publications -  122
Citations -  8321

Moriah E. Thomason is an academic researcher from New York University. The author has contributed to research in topics: Resting state fMRI & Medicine. The author has an hindex of 42, co-authored 99 publications receiving 6946 citations. Previous affiliations of Moriah E. Thomason include Stanford University & Harper University Hospital.

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Immature frontal lobe contributions to cognitive control in children: evidence from fMRI.

TL;DR: Children exhibited immature prefrontal activation that varied according to the type of cognitive control required, and were more susceptible to interference and less able to inhibit inappropriate responses than were adults.
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Default-mode and task-positive network activity in major depressive disorder: implications for adaptive and maladaptive rumination.

TL;DR: Findings support a formulation in which the DMN undergirds representation of negative, self-referential information in depression, and the RFIC, when prompted by increased levels of DMN activity, initiates an adaptive engagement of the TPN.
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Diffusion imaging, white matter, and psychopathology.

TL;DR: Brain changes that have been studied with DTI over the human lifespan and findings in a variety of neuropsychiatric disorders are reviewed and future areas where DTI is likely to have significant impact are suggested.
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Unraveling the Miswired Connectome: A Developmental Perspective

TL;DR: The most common mental illnesses can be conceptualized as developmental disorders of neural interactions within the connectome, or developmental miswiring as mentioned in this paper, and the recent maturation of pediatric in vivo brain imaging is bringing the identification of clinically meaningful brain-based biomarkers of developmental disorders within reach.
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Investigating neural primacy in Major Depressive Disorder: multivariate Granger causality analysis of resting-state fMRI time-series data.

TL;DR: It is shown that, on a moment-by-moment basis, there is increased excitatory activity among limbic and paralimbic structures, as well as increased inhibition in the activity of dorsal cortical structures, by limbic structures in depression.