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Sebastian Bartels

Researcher at University of Basel

Publications -  21
Citations -  1965

Sebastian Bartels is an academic researcher from University of Basel. The author has contributed to research in topics: Arabidopsis & Arabidopsis thaliana. The author has an hindex of 17, co-authored 21 publications receiving 1579 citations. Previous affiliations of Sebastian Bartels include University of Freiburg.

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The Arabidopsis bZIP transcription factor HY5 regulates expression of the; PFG1/MYB12; gene in response to light and ultraviolet-B radiation

TL;DR: The data suggest that HY5-dependent regulation of PFG gene expression contributes to the establishment of UV-B tolerance, and the bZIP transcriptional regulator ELONGATED HYPOCOTYL5 (HY5) is required for the transcriptional activation of the PFG1/MYB12 and PFG3/MYb111 genes underUV-B and visible light.
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MAP kinase phosphatase1 and protein tyrosine phosphatase1 are repressors of salicylic acid synthesis and SNC1-mediated responses in Arabidopsis

TL;DR: A major role of MKP1 and PTP1 is indicated in repressing salicylic acid biosynthesis in the autoimmune-like response caused by SNC1, which is largely the disease resistance gene homolog SUPPRESSOR of npr1-1, CONSTITUTIVE 1 that is absent in the Wassilewskija accession.
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Damage on plants activates Ca2+-dependent metacaspases for release of immunomodulatory peptides.

TL;DR: Pep1 generation was detected within 30 s after the injury, peaking at 5 min and lasting up to 1 hour in two wound model experiments on Arabidopsis seedlings, and Screening with various protease inhibitors revealed the involvement of metacaspases in Pep1 formation.
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Clathrin-dependent endocytosis is required for immunity mediated by pattern recognition receptor kinases

TL;DR: It is proposed that clathrin-dependent internalization of ligand-activated receptor kinases into a common endosomal pathway facilitates the responses required for full plant immunity, and that immunity mediated by pattern recognition receptors depends onClathrin, a critical component for the endocytosis of signaling competent receptors into acommon endosomes.
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Arabidopsis MAP kinase phosphatase 1 and its target MAP kinases 3 and 6 antagonistically determine UV‐B stress tolerance, independent of the UVR8 photoreceptor pathway

TL;DR: In this article, the authors show that loss of MAP kinase phosphatase 1 in the mutant MKp1 results in hypersensitivity to acute UV-B stress, but without impairing UV-b acclimation.