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Shuu Jiun Wang

Researcher at Taipei Veterans General Hospital

Publications -  544
Citations -  30138

Shuu Jiun Wang is an academic researcher from Taipei Veterans General Hospital. The author has contributed to research in topics: Migraine & Population. The author has an hindex of 71, co-authored 502 publications receiving 24800 citations. Previous affiliations of Shuu Jiun Wang include National Yang-Ming University & Taipei Medical University.

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Activation of neuropeptide Y Y1 receptors inhibits glutamate release through reduction of voltage-dependent Ca2+ entry in the rat cerebral cortex nerve terminals: Suppression of this inhibitory effect by the protein kinase C-dependent facilitatory pathway

TL;DR: The results suggest that NPY acting predominantly on NPY Y1 receptors inhibits glutamate release from rat cerebrocortical synaptosomes, likely by a mechanism involving direct coupling of receptors to N- and P/Q-type Ca2+ channels, and this coupling is subject to regulation by protein kinase C-dependent pathway.
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Hyperintense vessels: an early MRI marker of reversible cerebral vasoconstriction syndrome?

TL;DR: The unique finding that hyperintense vessels predated the occurrence of cerebral vasoconstrictions raised several important questions: 1.) CouldHyperintense vessel sign help diagnose RCVS in the very early stage of disease? 2.) Does this finding support the centripetal propagation theory? and 3.) How to differentiate hyperintensive vessels from cortical SAH, the early complication of RCVS.
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Cardiac cephalalgia. Case report and review of the literature with new ICHD-II criteria revisited.

TL;DR: A patient with cardiac cephalalgia is reported and reported cases from the English-language literature based on the new diagnostic criteria published in the International Classification of Headache Disorders, ed 2.
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Altered Brain Functional Connectome in Migraine with and without Restless Legs Syndrome: A Resting-State Functional MRI Study.

TL;DR: Findings suggest that migraine patients with and without RLS exhibit disruptions of brain FC that are associated with differential neuropathological mechanisms and may aid in the future development of neuroimaging-driven biomarkers for these conditions.