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Sowbiya Muneer

Researcher at VIT University

Publications -  66
Citations -  1731

Sowbiya Muneer is an academic researcher from VIT University. The author has contributed to research in topics: Biology & Photosynthesis. The author has an hindex of 19, co-authored 60 publications receiving 1117 citations. Previous affiliations of Sowbiya Muneer include Gyeongsang National University & Jamia Millia Islamia.

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Effect of CO, NOx and SO2 on ROS production, photosynthesis and ascorbate–glutathione pathway to induce Fragaria×annasa as a hyperaccumulator

TL;DR: The results indicate that increased ROS may act as a signal to induce defense responses to CO, NOx and SO2 gas stress, and the increased level of antioxidant enzymes plays a significant role in plant protection due to which strawberry plants can be used as a hyperaccumulator to maintain environmental pollution.
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Drought stress-induced physiological mechanisms, signaling pathways and molecular response of chloroplasts in common vegetable crops.

TL;DR: In this article, the chloroplast, organelle responsible for photosynthesis, is found to counteract the ill effects of drought stress by its critical involvement as a sensor of changes occurring in the environment, as the first process that drought stress affects is photosynthesis.
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Foliar or Subirrigation Silicon Supply Mitigates High Temperature Stress in Strawberry by Maintaining Photosynthetic and Stress-Responsive Proteins

TL;DR: The observed responses to silicon supply in high temperature stressed-plants indicate that Si, particularly in the form of K2SiO3, has a significant role in limiting the negative effects of high temperature stress by maintaining the photosynthetic proteins and stress-responsive proteins of the ascorbate glutathione defense mechanism.
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Involvement of sulphur nutrition in modulating iron deficiency responses in photosynthetic organelles of oilseed rape ( Brassica napus L.)

TL;DR: The present results indicate that S nutrition has significant role in ameliorating the damages in photosynthetic apparatus caused by Fe-deficiency.