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Stamatis-Nick C. Liossis
Researcher at University of Patras
Publications - 99
Citations - 4814
Stamatis-Nick C. Liossis is an academic researcher from University of Patras. The author has contributed to research in topics: Systemic lupus erythematosus & T cell. The author has an hindex of 33, co-authored 93 publications receiving 4243 citations. Previous affiliations of Stamatis-Nick C. Liossis include RMIT University & Uniformed Services University of the Health Sciences.
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Journal ArticleDOI
Experience with rituximab in scleroderma: results from a 1-year, proof-of-principle study
Dimitrios Daoussis,Stamatis-Nick C. Liossis,Athanassios C. Tsamandas,Christina Kalogeropoulou,Alexandra Kazantzi,Chaido Sirinian,Maria P. Karampetsou,Georgios Yiannopoulos,Andrew P. Andonopoulos +8 more
TL;DR: The results indicate that RTX may improve lung function in patients with SSc, and it is proposed that larger scale, multicentre studies with longer evaluation periods are needed.
Journal ArticleDOI
Pathogenesis of human systemic lupus erythematosus: recent advances
José C. Crispín,Stamatis-Nick C. Liossis,Katalin Kis-Toth,Linda A. Lieberman,Vasileios C. Kyttaris,Yuang-Taung Juang,George C. Tsokos +6 more
TL;DR: This work reviews recent findings that deal with genes associated with disease expression, immune cell molecular abnormalities that lead to autoimmune pathology, and the role of hormones and sex chromosomes in the development of disease.
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Altered pattern of TCR/CD3-mediated protein-tyrosyl phosphorylation in T cells from patients with systemic lupus erythematosus. Deficient expression of the T cell receptor zeta chain.
TL;DR: It is concluded that TCRzeta chain expression is either decreased or absent in the majority of patients with SLE, but not in patients with other systemic rheumatic diseases, regardless of disease activity, treatment status, or clinical manifestations.
Journal ArticleDOI
B cells from patients with systemic lupus erythematosus display abnormal antigen receptor-mediated early signal transduction events
TL;DR: This study demonstrates clearly for the first time that SLE B cells exhibit aberrant early signal transduction events, including augmented calcium responses after crosslinking of the B cell receptor and increased antigen-receptor-mediated phosphorylation of protein tyrosine residues.
Journal ArticleDOI
Evidence that Dkk‐1 is dysfunctional in ankylosing spondylitis
Dimitrios Daoussis,Stamatis-Nick C. Liossis,Elena E. Solomou,Anastasia Tsanaktsi,Konstadina Bounia,Maria P. Karampetsou,Georgios Yiannopoulos,Andrew P. Andonopoulos +7 more
TL;DR: The findings indicate that in patients with AS, circulating bone formation-promoting factors functionally prevail, which can be at least partially attributed to decreased Dkk-1-mediated inhibition.