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Stanley M. Hollenberg
Researcher at Fred Hutchinson Cancer Research Center
Publications - 6
Citations - 4209
Stanley M. Hollenberg is an academic researcher from Fred Hutchinson Cancer Research Center. The author has contributed to research in topics: Neural crest & NeuroD. The author has an hindex of 6, co-authored 6 publications receiving 4137 citations. Previous affiliations of Stanley M. Hollenberg include Oregon Health & Science University.
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Journal ArticleDOI
Mammalian Ras interacts directly with the serine/threonine kinase Raf
TL;DR: Raf interacts with wild-type and activated Ras, but not with an effector domain mutant of Ras or with a dominant-interfering Ras mutant, and this interaction is dependent on GTP bound to Ras.
Journal ArticleDOI
Conversion of Xenopus ectoderm into neurons by NeuroD, a basic helix-loop-helix protein
Jacqueline E. Lee,Stanley M. Hollenberg,Stanley M. Hollenberg,Lauren Snider,Lauren Snider,David L. Turner,Naomi Lipnick,Harold Weintraub,Harold Weintraub +8 more
TL;DR: The data suggest that neuroD may participate in the terminal differentiation step during vertebrate neuronal development and seems competent to bypass the normal inhibitory influences that usually prevent neurogenesis in ventral and lateral ectoderm.
Journal ArticleDOI
Identification of a new family of tissue-specific basic helix-loop-helix proteins with a two-hybrid system.
TL;DR: RNA in situ hybridization and reverse transcriptase-PCR demonstrate a stage- and tissue-specific distribution for the expression of Th1, a member of a new family of basic helix-loop-helix (bHLH) transcription factors with a reduced binding specificity.
Book ChapterDOI
Ras-Raf interaction: two-hybrid analysis.
TL;DR: This work has shown that the modified two-hybrid system has proved to be a powerful tool for identifying specific protein interactions, such as those between Ras and Raf, and hopes that the insight gained will prove valuable in the application of this system to other enigmatic questions in biology.
Journal ArticleDOI
Use of a conditional MyoD transcription factor in studies of MyoD trans-activation and muscle determination
TL;DR: Once muscle is induced by estrogen receptor-MyoD the muscle phenotype is very stable and does not need the continued presence of estradiol for its maintenance.