S
Stefan Hohmann
Researcher at Chalmers University of Technology
Publications - 205
Citations - 16932
Stefan Hohmann is an academic researcher from Chalmers University of Technology. The author has contributed to research in topics: Saccharomyces cerevisiae & Osmotic shock. The author has an hindex of 62, co-authored 204 publications receiving 15988 citations. Previous affiliations of Stefan Hohmann include University of the Free State & Technische Universität Darmstadt.
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A novel chloroplast localized Rab GTPase protein CPRabA5e is involved in stress, development, thylakoid biogenesis and vesicle transport in Arabidopsis
Sazzad Karim,Mohamed Alezzawi,Christel Garcia-Petit,Katalin Solymosi,Nadir Zaman Khan,Emelie Lindquist,Peter Dahl,Stefan Hohmann,Henrik Aronsson +8 more
TL;DR: A role of CPRabA5e in transport to and from thylakoids, similar to cytosolic Rab proteins involved in vesicle transport is suggested, consistent with the stress phenotypes observed.
Journal ArticleDOI
Yeast AMP-activated Protein Kinase Monitors Glucose Concentration Changes and Absolute Glucose Levels
Loubna Bendrioua,Maria Smedh,Joachim Almquist,Marija Cvijovic,Mats Jirstrand,Mattias Goksör,Caroline B. Adiels,Stefan Hohmann +7 more
TL;DR: The data suggest that the Snf1-Mig1 system has the ability to monitor glucose concentration changes as well as absolute glucose levels, and may provide for a highly flexible and fast adaptation to an altered nutritional status.
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Disruption of the Kluyveromyces lactis GGS1 gene causes inability to grow on glucose and fructose and is suppressed by mutations that reduce sugar uptake
K Luyten,W. de Koning,Ina Tesseur,M Ruiz,José Ramos,P Cobbaert,Johan M. Thevelein,Stefan Hohmann +7 more
TL;DR: In the yeast Saccharomyces cerevisiae the GGS1 gene is essential for growth on glucose or other readily fermentable sugars and short-term glucose-induced regulatory effects related to cAMP and cAMP-dependent protein kinase are studied.
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Glucose-induced regulatory defects in the Saccharomyces cerevisiae byp1 growth initiation mutant and identification of MIG1 as a partial suppressor.
TL;DR: Screening of a yeast gene library for clones complementing the byp1-3 phenotype resulted in the isolation of a truncated form of the previously described zinc finger transcription repressor MIG1, which suppressed even on a single-copy vector the growth initiation defect but not the regulatory abnormalities of the by p1- 3 mutant.