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Stefan Schmollinger

Researcher at University of California, Los Angeles

Publications -  37
Citations -  1199

Stefan Schmollinger is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Chlamydomonas & Chlamydomonas reinhardtii. The author has an hindex of 15, co-authored 30 publications receiving 851 citations. Previous affiliations of Stefan Schmollinger include Max Planck Society & California Institute for Quantitative Biosciences.

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Nitrogen-Sparing Mechanisms in Chlamydomonas Affect the Transcriptome, the Proteome, and Photosynthetic Metabolism

TL;DR: Comparison of the N-replete versus N-deplete proteome indicated that abundant proteins with a high N content are reduced in N-starved cells, while the proteins that are increased have lower than average N contents, suggesting an approach for engineering increased N-use efficiency.
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Multiomics resolution of molecular events during a day in the life of Chlamydomonas.

TL;DR: It is found that Chlamydomonas exhibits lower respiratory activity at night compared with the day; multiple fermentation pathways, some oxygen-sensitive, are expressed at night in aerated cultures; and it is proposed that the ferredoxin, FDX9, is potentially the electron donor to hydrogenases.
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An inducible artificial microRNA system for Chlamydomonas reinhardtii confirms a key role for heat shock factor 1 in regulating thermotolerance

TL;DR: This work equipped a recently developed amiRNA expression vector with the NIT1 promoter, which is repressed by ammonium and activated by nitrate, and tested this conditional ami RNA vector with heat shock factor 1 (HSF1) as target, supporting the earlier conclusion that HSF1 is a key regulator for thermotolerance in Chlamydomonas.
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Transcription Factor–Dependent Chromatin Remodeling at Heat Shock and Copper-Responsive Promoters in Chlamydomonas reinhardtii

TL;DR: Activated HSF1 and CRR1 transcription factors mediate the acetylation of histones H3/4, nucleosome eviction, remodeling of the H3K4 mono- and dimethylation marks, and transcription initiation/elongation at target genes, suggesting interplay of specific and presumably more generally acting factors to adapt gene expression to the new requirements of a changing environment.