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Stephen T. Chisholm

Researcher at Colorado State University

Publications -  15
Citations -  4704

Stephen T. Chisholm is an academic researcher from Colorado State University. The author has contributed to research in topics: Arabidopsis & Gene. The author has an hindex of 13, co-authored 15 publications receiving 4307 citations. Previous affiliations of Stephen T. Chisholm include University of California, Berkeley & Washington State University.

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Host-microbe interactions: Shaping the evolution of the plant immune response

TL;DR: In this review, taking an evolutionary perspective, important discoveries over the last decade about the plant immune response are highlighted.
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Gene amplification confers glyphosate resistance in Amaranthus palmeri

TL;DR: This work investigated recently discovered glyphosate-resistant Amaranthus palmeri populations from Georgia, in comparison with normally sensitive populations, and revealed that EPSPS genes were present on every chromosome and, therefore, gene amplification was likely not caused by unequal chromosome crossing over.
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Genetic and molecular evidence that the Pseudomonas syringae type III effector protein AvrRpt2 is a cysteine protease.

TL;DR: Results of a secondary structure prediction algorithm suggest that the functional C‐terminal portion of AvrRpt2 is a cysteine protease whose activity is required for elimination of RIN4 during infection.
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Cloning of the Arabidopsis RTM1 gene, which controls restriction of long-distance movement of tobacco etch virus

TL;DR: The locus RTM1 is necessary for restriction of long-distance movement of tobacco etch virus in Arabidopsis thaliana without causing a hypersensitive response or inducing systemic acquired resistance.
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Arabidopsis RTM2 gene is necessary for specific restriction of tobacco etch virus and encodes an unusual small heat shock-like protein.

TL;DR: Arabidopsis plants have a system to specifically restrict the long-distance movement of tobacco etch potyvirus without involving either hypersensitive cell death or systemic acquired resistance, and the RTM1/RTM2–mediated restriction was shown to be highly specific for TEV.