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Syeda Sadia Fatima

Researcher at Aga Khan University

Publications -  87
Citations -  3399

Syeda Sadia Fatima is an academic researcher from Aga Khan University. The author has contributed to research in topics: Diabetes mellitus & Gestational diabetes. The author has an hindex of 16, co-authored 79 publications receiving 1558 citations. Previous affiliations of Syeda Sadia Fatima include Bahria University.

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Global, regional, and national burden of chronic kidney disease, 1990–2017: a systematic analysis for the Global Burden of Disease Study 2017

Boris Bikbov, +221 more
- 29 Feb 2020 - 
TL;DR: The burden of CKD was much higher than expected for the level of development, whereas the disease burden in western, eastern, and central sub-Saharan Africa, east Asia, south Asia, central and eastern Europe, Australasia, and western Europe was lower than expected.
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Anti-Mullerian Hormone: Above and Beyond Conventional Ovarian Reserve Markers.

TL;DR: The role of AMH is summarized, it is proposed that AMH evaluation has a potential role in effectively monitoring chemotherapy and pelvic radiation induced ovarian toxicity, and potential clinical applications in children and adults are attempted.
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New roles of the multidimensional adipokine: chemerin.

TL;DR: Recent data of investigations on chemerin is consolidated in context to functional characteristics with a special reference to its role as a metabolic signal in inflammation and non-metabolic syndromes.
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Elevated chemerin levels in Pakistani men: an interrelation with metabolic syndrome phenotypes.

TL;DR: Serum chemerin levels were found to be independently and significantly associated with serum levels of cholesterol and glucose and may play a role in the development of insulin resistance, suggesting that chemer in may serve as an independent marker in diagnosing these conditions even before they become clinically symptomatic.
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Elevated levels of chemerin, leptin, and interleukin-18 in gestational diabetes mellitus

TL;DR: High chemerin and leptin levels are seen in GDM which may be associated with subclinical inflammation suggesting a role in development of insulin resistance.