T
Takeshi Yoshida
Researcher at University of Rochester Medical Center
Publications - 141
Citations - 4606
Takeshi Yoshida is an academic researcher from University of Rochester Medical Center. The author has contributed to research in topics: Lymphokine & Antigen. The author has an hindex of 36, co-authored 123 publications receiving 4104 citations. Previous affiliations of Takeshi Yoshida include State University of New York System & University at Buffalo.
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Journal ArticleDOI
Tight junction defects in patients with atopic dermatitis.
Anna De Benedetto,Nicholas Rafaels,Laura Y. McGirt,Andrei I. Ivanov,Steve N. Georas,Chris Cheadle,Alan E. Berger,Kunzhong Zhang,Sadasivan Vidyasagar,Takeshi Yoshida,Mark Boguniewicz,Tissa Hata,Lynda C. Schneider,Jon M. Hanifin,Richard L. Gallo,Natalija Novak,Stephan Weidinger,Terri H. Beaty,Donald Y.M. Leung,Kathleen C. Barnes,Lisa A. Beck +20 more
TL;DR: It is suggested that an impairment in tight junctions contributes to the barrier dysfunction and immune dysregulation observed in AD subjects and that this may be mediated in part by reductions in claudin-1.
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The cutaneous innate immune response in patients with atopic dermatitis.
TL;DR: The role of epidermal innate receptors in regulation of skin barrier is discussed in this article, where possible, discuss the relevance of these findings for patients with atopic dermatitis, where possible they discuss the importance of the epidermis in the treatment of skin infections and injury.
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The production of migration inhibition factor by b and t cells of the guinea pig
TL;DR: Although activation of lymphocytes for MIF production by specific antigen is a property of T cells, B cells as well as T cells may be so activated by agents which act nonspecifically, this may prove to have implications for in vivo events involved in immunization.
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Transcription Regulation of ompF and ompC by a Single Transcription Factor, OmpR
TL;DR: It is proposed that this tight hierarchical binding of a transcription factor, OmpR, allows distinct stepwise regulation of ompF and ompC transcription, which minimizes their overlapping expression upon changes in the medium osmolarity.
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Epidermal tight junctions in health and disease.
TL;DR: The preliminary observations suggesting that tight junction function or protein expression may be relevant for the pathogenesis of a number of common cutaneous inflammatory and neoplastic conditions are discussed.