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Tamami Homma

Researcher at Nagoya University

Publications -  8
Citations -  629

Tamami Homma is an academic researcher from Nagoya University. The author has contributed to research in topics: Kisspeptin & Luteinizing hormone. The author has an hindex of 7, co-authored 8 publications receiving 573 citations. Previous affiliations of Tamami Homma include University of Tsukuba.

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Possible Role of Oestrogen in Pubertal Increase of Kiss1/Kisspeptin Expression in Discrete Hypothalamic Areas of Female Rats

TL;DR: The present study suggests that a pubertal increase of Kiss1/kisspeptin expression both in the ARC and AVPV is involved in the onset of puberty, and suggests that both LH pulses and ARC Kiss1 expression are more negatively regulated by oestrogen in prepubertal female rats compared to adult rats.
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Molecular Characterization and Estrogen Regulation of Hypothalamic KISS1 Gene in the Pig

TL;DR: It is suggested that the PeN kisspeptin neurons could be responsible for the estrogen positive feedback regulation to induce gonadotropin-releasing hormone/luteinizing hormone (GnRH/LH) surge in the pig.
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Ultrastructural evidence of kisspeptin-gonadotrophin-releasing hormone (GnRH) interaction in the median eminence of female rats: implication of axo-axonal regulation of GnRH release.

TL;DR: The present results suggest that kisspeptin at least partly exerts stimulatory effects on GnRH release from the neuronal terminals of GnRH neurones by axo‐axonal nonsynaptic interaction in the median eminence.
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Physiological role of metastin/kisspeptin in regulating gonadotropin-releasing hormone (GnRH) secretion in female rats.

TL;DR: The lactating rat model provided further evidence indicating that ARC metastin/kisspeptin neurons are involved in GnRH pulse generation, because pulsatile release of luteinizing hormone (LH) is profoundly suppressed by suckling stimulus and the LH pulse suppression is well associated with the suppression of ARC metastIn/ Kisspeptin and KiSS-1 gene expression in lactating rats.