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Tara J. Holman

Researcher at University of Nottingham

Publications -  12
Citations -  1546

Tara J. Holman is an academic researcher from University of Nottingham. The author has contributed to research in topics: Arabidopsis thaliana & Germination. The author has an hindex of 11, co-authored 12 publications receiving 1331 citations. Previous affiliations of Tara J. Holman include Rothamsted Research.

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High-Throughput Quantification of Root Growth Using a Novel Image-Analysis Tool

TL;DR: A high-throughput, automatic method to trace Arabidopsis seedling roots grown on agarose plates that requires minimal interaction from the user and is able to process long time-lapse sequences with user interaction only required on the first frame.
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Seed after-ripening is a discrete developmental pathway associated with specific gene networks in Arabidopsis.

TL;DR: In this paper, the authors analyzed genome expression in Arabidopsis thaliana mutants defective in seed ABA synthesis (aba1-1) or perception (abi1 -1) and found that these mutants exhibited changes in global gene expression resulting from dry AR that were comparable with changes occurring in wild type (WT) seeds.
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The N-end rule pathway promotes seed germination and establishment through removal of ABA sensitivity in Arabidopsis

TL;DR: The N-end rule pathway targets protein degradation through the identity of the amino-terminal residue of specific protein substrates in Arabidopsis thaliana, and two components of this pathway, PROTEOLYSIS6 (PRT6) and arginyl-tRNA:protein arginially transferase (ATE), were shown to regulate seed after-ripening, seedling sugar sensitivity, seedlings lipid breakdown, and abscisic acid (ABA) sensitivity of germination as mentioned in this paper.
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Mathematical modeling elucidates the role of transcriptional feedback in gibberellin signaling

TL;DR: Integrated mathematical modeling and experiments are used to understand how feedback loops interact to control gibberellin signaling and find that regulation of GA20ox transcription plays a significant role in both modulating the level of endogenous gibbEREllin and generating overshoots after the removal of exogenous gibb Berellin.