T
Tariq Shafi
Researcher at University of Mississippi Medical Center
Publications - 149
Citations - 4161
Tariq Shafi is an academic researcher from University of Mississippi Medical Center. The author has contributed to research in topics: Kidney disease & Renal function. The author has an hindex of 31, co-authored 129 publications receiving 3175 citations. Previous affiliations of Tariq Shafi include University of Minnesota & Harper University Hospital.
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Journal ArticleDOI
Association of Residual Urine Output With Mortality, Quality of Life, and Inflammation in Incident Hemodialysis Patients: The Choices for Healthy Outcomes in Caring for End-Stage Renal Disease (CHOICE) Study
Tariq Shafi,Tariq Shafi,Bernard G. Jaar,Laura C. Plantinga,Nancy E. Fink,Nancy E. Fink,John H. Sadler,Rulan S. Parekh,Rulan S. Parekh,Rulan S. Parekh,Neil R. Powe,Neil R. Powe,Neil R. Powe,Josef Coresh +13 more
TL;DR: RKF in hemodialysis patients is associated with better survival and QOL, lower inflammation, and significantly less EPO use, and the development of methods to assess and preserve RKF is important and may improve dialysis care.
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Novel markers of kidney function as predictors of ESRD, cardiovascular disease, and mortality in the general population.
Brad C. Astor,Tariq Shafi,Ron C. Hoogeveen,Ron C. Hoogeveen,Kunihiro Matsushita,Christie M. Ballantyne,Christie M. Ballantyne,Lesley A. Inker,Josef Coresh +8 more
TL;DR: The addition of cystatin C, BTP, and B2M levels to models including eGFR(CKD-EPI) and all covariates, including urinary albumin-creatinine ratio, significantly improved risk prediction for all outcomes (P < 0.001).
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Trimethylamine N-Oxide and Cardiovascular Events in Hemodialysis Patients
Tariq Shafi,Neil R. Powe,Timothy W. Meyer,Seungyoung Hwang,Xin Hai,Michal L. Melamed,Tanushree Banerjee,Josef Coresh,Thomas H. Hostetter +8 more
TL;DR: It is concluded that TMAO concentration associates with cardiovascular events in hemodialysis patients but the effects differ by race, and whites in the highest quintile for T MAO had a 4-fold higher risk of cardiac or sudden cardiac death and a 2-foldHigher risk of any-cause death.
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Changes in Serum Potassium Mediate Thiazide-Induced Diabetes
TL;DR: Potassium supplementation might prevent thiazide-induced diabetes and appears to be mediated by changes in serum potassium, a hypothesis that can and should be tested in a randomized trial.
Journal Article
Changes in Serum Potassium Mediate Thiazide-Induced Diabetes. Commentary
TL;DR: In this paper, a randomized clinical trial of isolated systolic hypertension in individuals aged ≥60 years treated with chlorthalidone or placebo was conducted, and the results showed that the adjusted risk of diabetes was 2.07 (95% CI: 1.51 to 2.83; P<0.001).