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Teresa Rojo Romanos

Researcher at University of Copenhagen

Publications -  6
Citations -  80

Teresa Rojo Romanos is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Caenorhabditis elegans & Neuron. The author has an hindex of 5, co-authored 6 publications receiving 62 citations. Previous affiliations of Teresa Rojo Romanos include Dresden University of Technology & Monash University.

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EGL-13/SoxD specifies distinct O2 and CO2 sensory neuron fates in Caenorhabditis elegans.

TL;DR: The same core regulatory factor, egl-13, is required and sufficient to specify the distinct fates of O2- and CO2-sensing neurons in C. elegans.
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A Novel Role for the Zinc-Finger Transcription Factor EGL-46 in the Differentiation of Gas-Sensing Neurons in Caenorhabditis Elegans

TL;DR: EGL-46, a zinc-finger transcription factor, is identified, which regulates BAG gas-sensing fate in partially parallel pathways to ETS-5 and EGL-13, and is reported here the identification of three conserved transcription factors collaborate to ensure neuron type-specific identity features of the BAGGas-s sensing neurons.
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Control of Neuropeptide Expression by Parallel Activity-dependent Pathways in Caenorhabditis elegans.

TL;DR: Two parallel pathways regulate neuropeptide gene expression in the BAG sensory neurons: the ability to sense changes in carbon dioxide and CREB transcription factor and cAMP-dependent protein kinase (KIN-2) signaling pathway are shown.
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A heterodimer of evolved designer-recombinases precisely excises a human genomic DNA locus.

TL;DR: The generation of an SSR system that precisely excises a 1.4 kb fragment from the human genome is described, suggesting that designer-recombinases can be generated in a manageable timeframe for precision genome editing.
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LIN-32/Atonal Controls Oxygen Sensing Neuron Development in Caenorhabditis elegans.

TL;DR: The results show that aspects of URX neuronal fate are partially restored in lin-32 mutant animals when the apoptosis pathway is inhibited, suggesting that, as in other organisms, LIN-32/Atonal regulates neuronal apoptosis.