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Terry D. Etherton

Researcher at Pennsylvania State University

Publications -  103
Citations -  11824

Terry D. Etherton is an academic researcher from Pennsylvania State University. The author has contributed to research in topics: Adipose tissue & Insulin. The author has an hindex of 44, co-authored 103 publications receiving 11285 citations. Previous affiliations of Terry D. Etherton include University of Rochester Medical Center & United States Department of Agriculture.

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Bioactive compounds in foods: their role in the prevention of cardiovascular disease and cancer

TL;DR: Many bioactive compounds are extranutritional constituents that typically occur in small quantities in foods and are grouped accordingly as phenolic compounds, including their subcategory, flavonoids as discussed by the authors.
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Polyunsaturated fatty acids in the food chain in the United States

TL;DR: Alternative strategies, such as food enrichment and the use of biotechnology to manipulate the EPA and DHA as well as ALA contents of the food supply, will become increasingly important in increasing n-3 fatty acid intake in the US population.
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High-monounsaturated fatty acid diets lower both plasma cholesterol and triacylglycerol concentrations.

TL;DR: A high-MUFA, cholesterol-lowering diet may be preferable to a low-fat diet because of more favorable effects on the CVD risk profile.
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Effects of the National Cholesterol Education Program's Step I and Step II dietary intervention programs on cardiovascular disease risk factors: a meta-analysis

TL;DR: Step I and Step II dietary interventions have multiple beneficial effects on important cardiovascular disease risk factors and exercise resulted in greater decreases in TC, LDL cholesterol, and triacylglycerol and prevented the decrease in HDL cholesterol associated with low-fat diets.
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Dietary α-Linolenic Acid Reduces Inflammatory and Lipid Cardiovascular Risk Factors in Hypercholesterolemic Men and Women

TL;DR: ALA appears to decrease CVD risk by inhibiting vascular inflammation and endothelial activation beyond its lipid-lowering effects.