T
Teun J. de Vries
Researcher at Academic Center for Dentistry Amsterdam
Publications - 94
Citations - 2850
Teun J. de Vries is an academic researcher from Academic Center for Dentistry Amsterdam. The author has contributed to research in topics: Osteoclast & Bone resorption. The author has an hindex of 28, co-authored 79 publications receiving 2332 citations. Previous affiliations of Teun J. de Vries include VU University Amsterdam & University of Amsterdam.
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Journal ArticleDOI
Osteocytes subjected to fluid flow inhibit osteoclast formation and bone resorption.
S. Djien Tan,Teun J. de Vries,Anne Marie Kuijpers-Jagtman,C.M. Semeins,Vincent Everts,Jenneke Klein-Nulend +5 more
TL;DR: The osteocyte appears to be more responsive to PFF than the osteoblast or periosteal fibroblast regarding to the production of soluble factors affecting osteoclast formation and bone resorption.
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The epithelial Ca2+ channel TRPV5 is essential for proper osteoclastic bone resorption.
Bram C. J. van der Eerden,Joost G. J. Hoenderop,Teun J. de Vries,Ton Schoenmaker,C.J. Buurman,André G. Uitterlinden,Huibert A. P. Pols,René J. M. Bindels,Johannes P.T.M. van Leeuwen +8 more
TL;DR: TRPV5 deficiency leads to an increase in osteoclast size and number, in which Ca(2+) resorption is nonfunctional, and the significance of transcellular Ca( 2+) transport in osteoclastic function is demonstrated.
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Immune function and diversity of Osteoclasts in normal and pathological conditions
Maria-Bernadette Madel,Lidia Ibáñez,Abdelilah Wakkach,Teun J. de Vries,Anna Teti,Florence Apparailly,Claudine Blin-Wakkach +6 more
TL;DR: This review will address this novel vision of the OCL, not only as a phagocyte specialized in bone resorption, but also as innate immune cell participating in the control of immune responses.
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Transcription factor C/EBPβ isoform ratio regulates osteoclastogenesis through MafB
TL;DR: Disequilibrium between bone‐forming osteoblasts and bone‐resorbing osteoclasts is central to many bone diseases and data show that dysregulated expression of translationally controlled isoforms of CCAAT/enhancer‐binding protein β (C/EBPβ) differentially affect bone mass.
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Monocyte Heterogeneity: Consequences for Monocyte-Derived Immune Cells.
TL;DR: Recent findings in monocyte heterogeneity and their differentiation into dendritic cells, macrophages, and osteoclasts are summarized and advances in the context of the formation of functionally different monocyte-derived subsets of dendrite cells, Macrophage, and bone marrow cells are discussed.