Thomas S.K. Chang

TL;DR: No is established as a physiologic mediator of erectile function and small doses of NO synthase inhibitors abolished electrophysiologically induced penile erections.

TL;DR: Intraocular injections of RBZ provided benefit for patients with DME for at least 2 years, and when combined with focal or grid laser treatments, the amount of residual edema was reduced, as were the frequency of injections needed to control edema.

TL;DR: Nitric oxide synthase was identified in discrete neuronal locations, including the pelvic plexus, cavernous nerves and their terminal endings within the corporeal erectile tissue, branches of the dorsal penile nerves and nerve plexuses in the adventitia of the deep cavernous arteries, suggesting that nitric oxide neuronally modulates local vascular smooth musculature of the penis.

TL;DR: Nitric oxide synthase (NOS), which catalyzes the production of nitric oxide (NO), was characterized within the reproductive tract of adult male Sprague-Dawley rats by means of biochemical and immunohistochemical techniques.

TL;DR: In this article, the presence of N -nitro-larginine methyl ester (l-NAME) inhibitor in the male reproductive tract was confirmed by using the nitric oxide synthase-specific inhibitor.