T
Timothy S. Wallis
Researcher at Newbury College
Publications - 51
Citations - 5179
Timothy S. Wallis is an academic researcher from Newbury College. The author has contributed to research in topics: Salmonella enterica & Salmonella. The author has an hindex of 38, co-authored 51 publications receiving 4983 citations. Previous affiliations of Timothy S. Wallis include University of Giessen & University of Reading.
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Options for the control of enterohaemorrhagic Escherichia coli in ruminants
Journal ArticleDOI
SiiE Is Secreted by the Salmonella enterica Serovar Typhimurium Pathogenicity Island 4-Encoded Secretion System and Contributes to Intestinal Colonization in Cattle
TL;DR: It is reported that Salmonella enterica serovar Typhimurium pathogenicity island 4 carries a type I secretion system (siiCDF) which secretes an ∼600-kDa protein (encoded by siiE) which influences colonization in cattle and the invasion of bovine enterocytes.
Journal ArticleDOI
Current perspectives in salmonellosis.
TL;DR: Improved understanding of the pathogenesis of salmonellosis may also provide a model of wide applicability to a more general understanding of bacterial pathogenesis.
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EspP, a Type V-secreted serine protease of enterohaemorrhagic Escherichia coli O157:H7, influences intestinal colonization of calves and adherence to bovine primary intestinal epithelial cells.
Francis Dziva,Arvind Mahajan,Pamela Cameron,Carol G. Currie,Iain J. McKendrick,Timothy S. Wallis,David Smith,Mark P. Stevens +7 more
TL;DR: It is reported that the pO157-encoded Type V-secreted serine protease EspP influences the intestinal colonization of calves and adds to the growing body of evidence that cytotoxins facilitate intestinal colonization by EHEC.
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Net replication of Salmonella enterica serovars Typhimurium and Choleraesuis in porcine intestinal mucosa and nodes is associated with their differential virulence.
TL;DR: The rapid induction of proinflammatory responses by strain 4/74 may explain why pigs confine serovar Typhimurium infection to the intestines, whereas slow replication of Serovar Choleraesuis may enable it to evade host innate immunity and thus disseminate by stealth.