T
Toshio Onitsuka
Researcher at Yokohama National University
Publications - 130
Citations - 1875
Toshio Onitsuka is an academic researcher from Yokohama National University. The author has contributed to research in topics: Lung cancer & Cancer. The author has an hindex of 22, co-authored 130 publications receiving 1761 citations.
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Journal ArticleDOI
Distribution of Mast Cells in Mediastinal Lymph Nodes from Lung Cancer Patients
TL;DR: It is not possible to conclude whether mast cells in lymph nodes are for or against tumor spread, but an inverse relationship was observed between the number of mast cells and the amount of tumor tissue.
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Goblet cell hyperplasia in the airway of Nippostrongylus brasiliensis-infected rats.
TL;DR: Airway goblet cell hyperplasia observed at days 14 and 21 after N. brasiliensis infection may be induced by local pulmonary factors, indicating that airwayGoblet cells have characteristics that differ from those of the small intestine.
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Expression of nm23-H1 gene product in esophageal squamous cell carcinoma and its association with vessel invasion and survival
Masaki Tomita,Takanori Ayabe,Yasunori Matsuzaki,Masao Edagawa,Masayuki Maeda,Tetsuya Shimizu,Masaki Hara,Toshio Onitsuka +7 more
TL;DR: In patients with esophageal squamous cell carcinoma, there appears to be an inverse relationship between nm23-H1 gene product expression and lymphatic vessel invasion, and this might be a prognostic marker in patients with involved lymph nodes.
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Spleen plays an important role in maintaining tolerance after removal of the vascularized heart graft.
Eiichi Chosa,Masaki Hara,Akira Watanabe,Yasunori Matsuzaki,Kunihide Nakamura,Kimikazu Hamano,Kathryn J. Wood,Toshio Onitsuka +7 more
TL;DR: The spleen is important in maintaining CD4+CD25+ regulatory T cells after primary allograft removal, as depleting anti-CD25 treatment in tolerant mice abrogated graft prolongation when spleen leukocytes were adoptively transferred to syngeneic mice.
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Reduction of post-ischemic lung reperfusion injury by fibrinolytic activity suppression.
Masao Edagawa,Etsuo Yoshida,Yasunori Matsuzaki,Kohji Shibuya,Koichiro Shibata,Toshio Onitsuka,Masugi Maruyama +6 more
TL;DR: The findings suggest that the fibrinolytic system is involved in the onset mechanism of ischemia-reperfusion injury through induced endothelial cell damage and increased vascular permeability.