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Yukifumi Nawa

Researcher at Khon Kaen University

Publications -  300
Citations -  6987

Yukifumi Nawa is an academic researcher from Khon Kaen University. The author has contributed to research in topics: Paragonimiasis & Nippostrongylus brasiliensis. The author has an hindex of 42, co-authored 299 publications receiving 6587 citations. Previous affiliations of Yukifumi Nawa include Seoul National University & Autonomous University of Sinaloa.

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The current status of opisthorchiasis and clonorchiasis in the Mekong Basin.

TL;DR: It is disturbing that despite treatment and control programs in place for decades, all countries of the Lower Mekong Basin are still highly endemic with O. viverrini and/or C. sinensis as well as alarmingly high levels of CCA incidence.
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Selective effector mechanisms for the expulsion of intestinal helminths.

TL;DR: By means of simple experimental approaches, it is found that mucosal mast cells and goblet cells are in fact highly selective and specific effector cells of the host defence mechanisms capable of acting on the establishment and the expulsion of intestinal helminths.
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Adventitial Mast Cells Contribute to Pathogenesis in the Progression of Abdominal Aortic Aneurysm

TL;DR: In this article, the authors carried out histological study of abdominal aortic aneurysm (AAA) tissues of patients, and interventional animal and cell culture experiments to investigate a role of mast cells in the pathogenesis of AAA.
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Drug resistance in pathogenic African trypanosomes: what hopes for the future?

TL;DR: It is the opinion that for now the management and control of trypanosomosis will continue to depend on proper usage of the few available trypanocides, especially strategic deployment of the sanative drugs in order to reduce the development of drug resistance, in addition to the continued use of environmentally friendly vector control programmes such tsetse trapping.
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Mucosal T Cells Bearing TCRγδ Play a Protective Role in Intestinal Inflammation

TL;DR: Transfer of γδ IEL to Cδ−/− mice ameliorated TNBS-induced colitis, which correlated with decrease of IFN-γ and TNF-α production and an increase of TGF-β production by IEL, and suggests that enhancement of regulatory γ δ T cell activity is a possible new cell therapy for colitis.