V
Vaishali R. Moulton
Researcher at Beth Israel Deaconess Medical Center
Publications - 44
Citations - 2240
Vaishali R. Moulton is an academic researcher from Beth Israel Deaconess Medical Center. The author has contributed to research in topics: T cell & Autoimmunity. The author has an hindex of 19, co-authored 42 publications receiving 1389 citations. Previous affiliations of Vaishali R. Moulton include University of Maryland, Baltimore.
Papers
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Journal ArticleDOI
Sex Hormones in Acquired Immunity and Autoimmune Disease.
TL;DR: The role of sex hormones particularly estrogen, in the adaptive immune response, in health, and autoimmune disease with an emphasis on systemic lupus erythematosus is focused on.
Journal ArticleDOI
Pathogenesis of Human Systemic Lupus Erythematosus: A Cellular Perspective
Vaishali R. Moulton,Abel Suárez-Fueyo,Esra Meidan,Esra Meidan,Hao Li,Masayuki Mizui,George C. Tsokos +6 more
TL;DR: Novel observations have provided an improved understanding of the contribution of tissue-specific factors and associated damage, T and B lymphocytes, as well as innate immune cell subsets and their corresponding abnormalities.
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Aging, Immunity, and COVID-19: How Age Influences the Host Immune Response to Coronavirus Infections?
Varnica Bajaj,Varnica Bajaj,Nirupa Gadi,Nirupa Gadi,Allison P. Spihlman,Allison P. Spihlman,Samantha C. Wu,Samantha C. Wu,Christopher H. Choi,Christopher H. Choi,Vaishali R. Moulton +10 more
TL;DR: In this article, age-related quantitative and qualitative changes in the immune system affect cells and soluble mediators of both the innate and adaptive immune responses within lymphoid and non-lymphoid peripheral tissues.
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T cell signaling abnormalities contribute to aberrant immune cell function and autoimmunity
TL;DR: A better understanding of signaling and gene regulation defects in SLE T cells will lead to the identification of specific novel molecular targets and predictive biomarkers for therapy.
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Abnormalities of T cell signaling in systemic lupus erythematosus
TL;DR: Systemic lupus erythematosus (SLE) is an autoimmune disease resulting from a loss of tolerance to multiple self antigens, and characterized by autoantibody production and inflammatory cell infiltration in target organs, such as the kidneys and brain.