V
Vangipuram Dwarakanath
Researcher at University of Texas Southwestern Medical Center
Publications - 24
Citations - 1188
Vangipuram Dwarakanath is an academic researcher from University of Texas Southwestern Medical Center. The author has contributed to research in topics: Sodium–hydrogen antiporter & Hypophosphatemia. The author has an hindex of 17, co-authored 24 publications receiving 1107 citations. Previous affiliations of Vangipuram Dwarakanath include University of Texas at Dallas.
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Journal ArticleDOI
FGF23 decreases renal NaPi-2a and NaPi-2c expression and induces hypophosphatemia in vivo predominantly via FGF receptor 1.
Jyothsna Gattineni,Carlton M. Bates,Katherine Twombley,Vangipuram Dwarakanath,Michael L. Robinson,Regina Goetz,Moosa Mohammadi,Michel Baum +7 more
TL;DR: Data show that FGFR1 is the predominant receptor for the hypophosphatemic action of FGF23 in vivo, with FGFR4 likely playing a minor role.
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Effect of fibroblast growth factor-23 on phosphate transport in proximal tubules
TL;DR: It is demonstrated that the inhibition of phosphate transport by FGF23R176Q in vitro requires heparin and is associated with a reduction in BBMV NaPi-2A protein abundance.
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Effect of renal denervation on prenatal programming of hypertension and renal tubular transporter abundance
TL;DR: This study is consistent with renal nerves playing a role in mediating the hypertension by prenatal programming by dexamethasone.
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Glucocorticoids acutely increase cell surface Na+/H+ exchanger-3 (NHE3) by activation of NHE3 exocytosis
I. Alexandru Bobulescu,Vangipuram Dwarakanath,Lixian Zou,Jianning Zhang,Michel Baum,Orson W. Moe +5 more
TL;DR: Exocytosis is the major contributor to the glucocorticoid-induced rapid increase in cell surface NHE3 protein abundance and Na(+)/H(+) exchange activity, which suggests that trafficking plays a key role in the acute stimulation of N HE3 by glucoc Corticoids.
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Effects of thyroid hormone on the neonatal renal cortical Na+/H+ antiporter
TL;DR: Data are consistent with a potential role of thyroid hormone in the postnatal increase in Na+/H+ antiporter activity.