Effects of thyroid hormone on the neonatal renal cortical Na+/H+ antiporter
TLDR
Data are consistent with a potential role of thyroid hormone in the postnatal increase in Na+/H+ antiporter activity.About:
This article is published in Kidney International.The article was published on 1998-05-01 and is currently open access. It has received 70 citations till now. The article focuses on the topics: Euthyroid & Sodium–hydrogen antiporter.read more
Citations
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Journal ArticleDOI
FGF23 decreases renal NaPi-2a and NaPi-2c expression and induces hypophosphatemia in vivo predominantly via FGF receptor 1.
Jyothsna Gattineni,Carlton M. Bates,Katherine Twombley,Vangipuram Dwarakanath,Michael L. Robinson,Regina Goetz,Moosa Mohammadi,Michel Baum +7 more
TL;DR: Data show that FGFR1 is the predominant receptor for the hypophosphatemic action of FGF23 in vivo, with FGFR4 likely playing a minor role.
Journal ArticleDOI
The Renal Manifestations of Thyroid Disease
TL;DR: Thyroid hormones influence renal development, kidney structure, renal hemodynamics, GFR, the function of many transport systems along the nephron, and sodium and water homeostasis.
Journal ArticleDOI
Interactions between thyroid disorders and kidney disease
Gopal Basu,Anjali Mohapatra +1 more
TL;DR: There are several interactions between thyroid and kidney functions in each other organ's disease states and a detailed knowledge of all these interactions is important for both the nephrologists and endocrinologists for optimal management of the patient.
Journal ArticleDOI
Rapid Nongenomic Effects of 3,5,3′-Triiodo-l-Thyronine on the Intracellular pH of L-6 Myoblasts Are Mediated by Intracellular Calcium Mobilization and Kinase Pathways
Silvia D'Arezzo,Sandra Incerpi,Faith B. Davis,Filippo Acconcia,Maria Marino,Ricardo N. Farías,Paul J. Davis +6 more
TL;DR: It is shown here for the first time that transduction of the hormone signal in this nongenomic response requires tyrosine kinase-dependent phospholipase C activation and two different signaling pathways: mobilization of intracellular calcium and protein phosphorylation involving protein kinase C and MAPK (ERK1/2).
Journal ArticleDOI
Thyroid dysfunction and kidney disease: An update
TL;DR: Interpretation of the interactions between thyroid and renal function is a challenge for clinicians involved in the treatment of patients with thyroid and kidney disease.
References
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Journal ArticleDOI
Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction
TL;DR: A new method of total RNA isolation by a single extraction with an acid guanidinium thiocyanate-phenol-chloroform mixture is described, providing a pure preparation of undegraded RNA in high yield and can be completed within 4 h.
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Maternal and Fetal Thyroid Function
TL;DR: In this article, recent data and new insights regarding the changes in thyroid metabolism are reviewed and the role of the placenta and amniotic fluid in these changes is reviewed.
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Molecular cloning of putative members of the Na/H exchanger gene family. cDNA cloning, deduced amino acid sequence, and mRNA tissue expression of the rat Na/H exchanger NHE-1 and two structurally related proteins.
TL;DR: Tse et al. as discussed by the authors used NHE-1 and NHE2 cDNAs to identify the functional diversity of the Na/H exchanger in mammals based on expression of multiple members of a gene family.
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Plasma concentrations of total and free corticosterone during development in the rat.
TL;DR: The data suggest that the rise in free corticosterone that begins on day 14 acts as a cue for enzymic changes in both liver and intestine.
Journal ArticleDOI
Expression of NHE-3 in the apical membrane of rat renal proximal tubule and thick ascending limb
Morimasa Amemiya,Jan Loffing,Marius Lötscher,Brigitte Kaissling,Robert J. Alpern,Orson W. Moe +5 more
TL;DR: NHE-3 is the isoform responsible for NaCl and NaHCO3 absorption in the proximal convoluted tubule, and NahCO absorption in a thick ascending limb and apical membrane Na/H exchange activity is likely mediated by other isoform of the NHE family.
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Development of solute transport in rabbit proximal tubule. I. HCO-3 and glucose absorption.
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