V
Vijay Yajnik
Researcher at Harvard University
Publications - 111
Citations - 6272
Vijay Yajnik is an academic researcher from Harvard University. The author has contributed to research in topics: Inflammatory bowel disease & Crohn's disease. The author has an hindex of 33, co-authored 105 publications receiving 5334 citations. Previous affiliations of Vijay Yajnik include Cleveland Clinic & Columbia University Medical Center.
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Journal ArticleDOI
Correlations Between Clinical Activity Indices or Quality of Life Scores to Endoscopic Activity Scores Do not Vary According to Age at Diagnosis, Disease Location, and Disease Behavior in Inflammatory Bowel Disease: 1883
Sasha Taleban,Kathleen O. Stewart,Darrick K. Li,Prashant Singh,Holly C. Sturgeon,Deanna Nguyen,Vijay Yajnik,Ashwin N. Ananthakrishnan,Hamed Khalili +8 more
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DOCK4 Regulation of Rho GTPases Mediates Pulmonary Vascular Barrier Function
Pascal Yazbeck,Xavier Cullere,Paul Bennett,Vijay Yajnik,Huan Wang,Kenji Kawada,Vanessa Davis,Asit Parikh,Andrew Kuo,Vijayashree Mysore,Timothy Hla,David S. Milstone,Tanya N. Mayadas +12 more
TL;DR: The findings indicate that DOCK4 maintains AJs necessary for lung vascular barrier function by establishing the normal balance between RhoA (Ras homolog family member A) and Rac-1–mediated actin cytoskeleton remodeling, a previously unappreciated function for the atypical GEF family of molecules.
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Systematic Review and Meta-Analysis: Risk of Colorectal Cancer in Patients of Ulcerative Colitis and an Asian Perspective
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Sa1235 PKM2 As a Novel Serum Biomarker of IBD Activity, A Early Pilot Study
Romela Marin,Hamed Khalili,Ashwin N. Ananthakrishnan,Jenny Sauk,Vijay Yajnik,Nitin Gupta,Ramnik J. Xavier,Deanna D. van Nguyen +7 more
TL;DR: TNF-α levels strongly correlated with disease activity based on HBI and CDEIS indices in patients with CD in maintenance treatment with ADA and correlated them with clinical and endoscopic disease activity.
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Mo1754 Early Life Environment Interacts With Genetic Risk in Inflammatory Bowel Diseases
Ashwin N. Ananthakrishnan,Jenny Sauk,Deanna D. Nguyen,Hamed Khalili,Joshua R. Korzenik,Vijay Yajnik,Hailiang Huang,Mark J. Daly,Ramnik J. Xavier +8 more
TL;DR: It is suggested that AIEC bacteria have the ability to modulate gene expression in the host cell for their own benefit by altering different epigenetic marks as miRNA expression profile and histones post-translational modifications enhancing CEACAM6 abnormal expression.