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Vivek Mishra

Researcher at Council of Scientific and Industrial Research

Publications -  36
Citations -  732

Vivek Mishra is an academic researcher from Council of Scientific and Industrial Research. The author has contributed to research in topics: Pancreatitis & Skeletal muscle. The author has an hindex of 13, co-authored 34 publications receiving 619 citations. Previous affiliations of Vivek Mishra include Mayo Clinic & Academy of Scientific and Innovative Research.

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The E3 ubiquitin ligase TRAF6 intercedes in starvation-induced skeletal muscle atrophy through multiple mechanisms.

TL;DR: The role of tumor necrosis factor receptor-associated factor 6 (TRAF6), a unique E3 ubiquitin ligase and adaptor protein, in starvation-induced muscle atrophy is unveiled and lysine 63-linked autoubiquitination of TRAF6 is identified as a process essential for its regulatory role in starved Muscle atrophy.
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Fibrosis reduces severity of acute-on-chronic pancreatitis in humans.

TL;DR: Based on analysis of pancreatic samples from patients with CP, AP, or AP-on-CP and in vitro studies, fibrosis reduces the severity of acute exacerbations of CP by reducing lipolytic flux between adipocytes and acinar cells.
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Regulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program

TL;DR: It is demonstrated that TWEAK significantly reduces the levels of PGC‐1α and mitochondrial content in skeletal muscle and induces muscle atrophy through repressing the Levels of P GC‐1a.
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Withania somnifera improves semen quality by combating oxidative stress and cell death and improving essential metal concentrations.

TL;DR: It is concluded that W. somnifera improves semen quality by reducing oxidative stress and cell death and improving essential metal ion concentrations in infertile subjects.
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Proinflammatory Cytokine Tumor Necrosis Factor (TNF)-like Weak Inducer of Apoptosis (TWEAK) Suppresses Satellite Cell Self-renewal through Inversely Modulating Notch and NF-κB Signaling Pathways

TL;DR: It is demonstrated that TWEAK cytokine reduces the proportion of Pax7+/MyoD− cells (an index of self-renewal) on myofiber explants and represses multiple components of Notch signaling in satellite cell cultures and a reciprocal interaction between NF-κB and NotCh signaling governs the inhibitory effect of T WEAK on satellite cell self-Renewal.