Wangquan Ji

TL;DR: The present understanding of COVID-19 is detailed and the current state of development of measures are introduced in this review to provide a comprehensive summary to public health authorities and potential readers worldwide.

TL;DR: In this paper, the proposed cellular and molecular mechanisms of endothelial activation and dysfunction during the outbreak of coronavirus disease 2019 (COVID-19) emphasizing the principal mediators and therapeutic implications.

TL;DR: In this paper, the authors identified three CVA2 strains from hand, foot, and mouth disease (HFMD) infections and used the cell-adapted coxsackievirus (CV) A2 strain HN202009 to inoculate 5-day-old BALB/c mice intramuscularly.

TL;DR: In this article , the expression of ACE2 and ACE and activation of inflammatory signaling pathways in lung tissues were evaluated by immunofluorescence staining and Western blotting, and it was found that PM2.5 exposure increased ACE2 expression significantly elevated the levels of total proteins, total cells, and the concentrations of MCP-1, IL-1β in bronchoalveolar lavage fluid (BALF).

TL;DR: In this article, the authors used the 5-day-old BALB/c mouse model of lethal CVA2 infection to evaluate lung damage, and found that the permeability of lung microvascular was significantly increased after the infection.