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Wayne S. Cutfield

Researcher at University of Auckland

Publications -  271
Citations -  10389

Wayne S. Cutfield is an academic researcher from University of Auckland. The author has contributed to research in topics: Pregnancy & Insulin resistance. The author has an hindex of 49, co-authored 248 publications receiving 9012 citations. Previous affiliations of Wayne S. Cutfield include Zhejiang University & Gravida.

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Premature birth and later insulin resistance.

TL;DR: Like children who were born at term but who were small for gestational age, children who had been born prematurely have an isolated reduction in insulin sensitivity, which may be a risk factor for type 2 diabetes mellitus.
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Neonatal Leptin Treatment Reverses Developmental Programming

TL;DR: The complete normalization of the programmed phenotype by neonatal leptin treatment implies that leptin has effects that reverse the prenatal adaptations resulting from relative fetal undernutrition, and is potentially reversible by an intervention late in the phase of developmental plasticity.
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Insulin resistance in short children with intrauterine growth retardation.

TL;DR: Short prepubertal IUGR children have a specific impairment in insulin sensitivity compared to their normal birth weight peers, a potential marker for the early identification and intervention in the development of late adult-onset noninsulin-dependent diabetes mellitus.
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Insulin Resistance in Children: Consensus, Perspective, and Future Directions

TL;DR: There are no clear criteria to define insulin resistance in children, and surrogate markers such as fasting insulin are poor measures of insulin sensitivity, so based on current screening criteria and methodology, there is no justification for screening children for insulin resistance.
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The fetal, neonatal, and infant environments-the long-term consequences for disease risk.

TL;DR: It is suggested that greater disease risk is created by a mismatch between the environment predicted during the plastic phase of development and the actual environment experienced in the postplastic phase.