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Wei Wang

Researcher at Nanjing Medical University

Publications -  9
Citations -  971

Wei Wang is an academic researcher from Nanjing Medical University. The author has contributed to research in topics: Computer science & Chemistry. The author has an hindex of 5, co-authored 5 publications receiving 889 citations. Previous affiliations of Wei Wang include Huazhong University of Science and Technology.

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Regulated ATP release from astrocytes through lysosome exocytosis

TL;DR: It is found that lysosomes contain abundant ATP, which could be released in a stimulus-dependent manner and implicating physiological and pathological functions of regulated lysOSome exocytosis in these cells.
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Neuroprotective effect of curcumin on focal cerebral ischemic rats by preventing blood-brain barrier damage.

TL;DR: It is concluded that curcumin ameliorates cerebral ischemia/reperfusion injury by preventing ONOO(-) mediated blood-brain barrier damage.
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Voluntary exercise-induced neurogenesis in the postischemic dentate gyrus is associated with spatial memory recovery from stroke

TL;DR: Exposure to voluntary exercise after ischemic stroke may promote newborn cells survival in the dentate gyrus by up‐regulating CREB phosphorylation and consequently restore impaired hippocampus‐dependent memory in adult mice.
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Reduced neuronal nitric oxide synthase is involved in ischemia-induced hippocampal neurogenesis by up-regulating inducible nitric oxide synthase expression.

TL;DR: It is suggested that reduced nNOS is involved in ischemia‐induced hippocampal neurogenesis by up‐regulating iNOS expression and cAMP response element‐binding protein phosphorylation.
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Blockade of L‐type voltage‐gated Ca2+ channel inhibits ischemia‐induced neurogenesis by down‐regulating iNOS expression in adult mouse

TL;DR: It is shown that mice subjected to 90 min of middle cerebral artery occlusion (MCAO) significantly increased the number of new neurons and up‐regulated iNOS expression in the dentate gyrus and down‐regulation of Ca2+ influx through L‐VGCC is involved in ischemia‐induced neurogenesis by up‐regulating iN OS expression.