W
Wenhao Zhou
Researcher at Boston Children's Hospital
Publications - 219
Citations - 6730
Wenhao Zhou is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 33, co-authored 168 publications receiving 4815 citations. Previous affiliations of Wenhao Zhou include Chinese Ministry of Health & Center for Excellence in Education.
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Vertical Transmission of Severe Acute Respiratory Syndrome Coronavirus 2 From the Mother to the Infant-Reply.
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Data on mutations and Clinical features in SCN1A or SCN2A gene.
Yanting Kong,Kai Yan,Liyuan Hu,Mingbang Wang,Xinran Dong,Yulan Lu,Bingbing Wu,Huijun Wang,Lin Yang,Wenhao Zhou +9 more
TL;DR: This dataset presented variants and clinical features of SCN1A and SCN2A genes and can provide insights on novel mutations and different phenotypes ofSCN1a and SCn2A.
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Frequent mutation of hypoxia-related genes in persistent pulmonary hypertension of the newborn.
Mingbang Wang,Deyi Zhuang,Mei Mei,Haiyan Ma,Zixiu Li,Fusheng He,Guoqiang Cheng,Guang Lin,Wenhao Zhou +8 more
TL;DR: The results indicates that the association of hypoxia-related genes with P PHN does not depend on high-altitude life, and 21 rare mutations associated with PPHN were found, including three rare variants of the tubulin tyrosine ligase-like family member 3 gene
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Genetic Spectrum Identified by Exome Sequencing in a Chinese Pediatric Cerebral Palsy Cohort.
Hong-Fang Mei,Alessandra Fabiane Lago,Lin Yang,Tiantian Xiao,Sujuan Wang,Bingbing Wu,Huijun Wang,Yulan Lu,Xinran Dong,Hong Yang,Wenhao Zhou +10 more
TL;DR: Wang et al. as discussed by the authors explored the genetic spectrum of cerebral palsy (CP) in a Chinese paediatric cohort and found that patients without any clinical risk factors or with a family history were more likely to have genetic risk factors.
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Mutations of CNTNAP1 led to defects in neuronal development.
Wanxing Li,Lin Yang,Chuanqing Tang,Kaiyi Liu,Yulan Lu,Huijun Wang,Kai Yan,Zilong Qiu,Wenhao Zhou +8 more
TL;DR: It is found that Cntnap1 is highly expressed in neurons and is located predominantly in MAP2+ neurons during the early developmental stage, providing insights underlying molecular and circuit mechanisms of CNTNAP1-related disease.