W
Wolfgang F. Graier
Researcher at Medical University of Graz
Publications - 256
Citations - 11197
Wolfgang F. Graier is an academic researcher from Medical University of Graz. The author has contributed to research in topics: Mitochondrion & Endoplasmic reticulum. The author has an hindex of 55, co-authored 236 publications receiving 9855 citations. Previous affiliations of Wolfgang F. Graier include University of Missouri & University of Hong Kong.
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N-acetyltransferase 8-like accelerates lipid turnover and increases energy in brown adipocytes
Ariane Pessentheiner,Helmut Josef Pelzmann,Evelyn Walenta,Martina Schweiger,Lukas N. Groschner,Wolfgang F. Graier,Dagmar Kolb,Kyosuke Uno,Toh Miyazaki,Atsumi Nitta,Dietmar Rieder,Andreas Prokesch,Juliane Gertrude Bogner-Strauß +12 more
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Kisspeptin-10, a KiSS-1/metastin-derived decapeptide, is a physiological invasion inhibitor of primary human trophoblasts.
Martin Bilban,Martin Bilban,Martin Bilban,Nassim Ghaffari-Tabrizi,Edith Hintermann,Sandra Bauer,Sylvia Molzer,Cristina Zoratti,Roland Malli,Andrew B. Sharabi,Ursula Hiden,Wolfgang F. Graier,Martin Knöfler,Fritz Andreae,Oswald Wagner,Vito Quaranta,Gernot Desoye +16 more
TL;DR: Kp-10, a dekapeptide derived from the primary translation product of KiSS-1, was identified as a novel paracrine/endocrine regulator in fine-tuning trophoblast invasion generated by the trophoblasts itself.
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Uncoupling proteins 2 and 3 are fundamental for mitochondrial Ca2+ uniport.
TL;DR: It is demonstrated that UCP2 and UCP3 are elementary for mitochondrial Ca(2+) sequestration in response to cell stimulation under physiological conditions - observations supported by isolated liver mitochondria of Ucp2(-/-) mice lacking ruthenium red-sensitive Ca( 2+) uptake.
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Coassembly of Trp1 and Trp3 proteins generates diacylglycerol- and Ca2+-sensitive cation channels.
Birgit Lintschinger,Monika Balzer-Geldsetzer,Tyagarajan Baskaran,Wolfgang F. Graier,Christoph Romanin,Michael X. Zhu,Klaus Groschner +6 more
TL;DR: It is concluded that coassembly of Trp1 and Trp3 resulted in the formation of oligomeric Trp channels that are subject to regulation by phospholipase C and Ca(2+).
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Cytochrome P450 mono-oxygenase-regulated signalling of Ca2+ entry in human and bovine endothelial cells.
TL;DR: The hypothesis that depletion of endothelial Ca2+ stores activates microsomal P450 MO which in turn synthesizes 5,6‐EET is supported, and it is proposed that the arachidonic acid metabolite 5, 6‐ EET or one of its metabolites is a second messenger for activation of endothelium‐derived nitric oxide (EDNO) in vascular endothelial cells.