W
Woo-Jong Kim
Researcher at Pohang University of Science and Technology
Publications - 9
Citations - 351
Woo-Jong Kim is an academic researcher from Pohang University of Science and Technology. The author has contributed to research in topics: Virus & Micropatterning. The author has an hindex of 5, co-authored 9 publications receiving 249 citations.
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Journal ArticleDOI
Mutations in DDX58, which Encodes RIG-I, Cause Atypical Singleton-Merten Syndrome
Mi-Ae Jang,Eun Kyoung Kim,Hesung Now,Nhung Thi-Cam Nguyen,Woo-Jong Kim,Joo-Yeon Yoo,Jinhyuk Lee,Jinhyuk Lee,Yun-Mi Jeong,Cheol-Hee Kim,Ok Hwa Kim,Seongsoo Sohn,Seong-Hyeuk Nam,Yoojin Hong,Yong-seok Lee,Sung-A Chang,Shin Yi Jang,Jong-Won Kim,Myung-Shik Lee,So Young Lim,Ki-Sun Sung,Ki-Tae Park,Byoung Joon Kim,Joo-Heung Lee,Duk-Kyung Kim,Changwon Kee,Chang-Seok Ki +26 more
TL;DR: It is demonstrated that DDX58 mutations cause atypical SMS manifesting with variable expression of glaucoma, aortic calcification, and skeletal abnormalities without dental anomalies.
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Freeform micropatterning of living cells into cell culture medium using direct inkjet printing.
Ju An Park,Sejeong Yoon,Jimin Kwon,Hesung Now,Young Kwon Kim,Woo-Jong Kim,Joo-Yeon Yoo,Sungjune Jung +7 more
TL;DR: This work presents a drop-on-demand inkjet printing method to directly pattern living cells into a cell-friendly liquid environment and applies this technique to investigate the influence of the heterogeneity and geometry of the cell population on the infectivity of seasonal H1N1 influenza virus.
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Ubiquitin-like modifier FAT10 attenuates RIG-I mediated antiviral signaling by segregating activated RIG-I from its signaling platform.
TL;DR: This study presented a novel mechanism to dampen RIG-I activity, which uncovered the suppressive effect of the accumulated FAT10 during virus-mediated inflammatory response and may also provide molecular clue to understand the carcinogenesis related with infection and inflammation.
Journal ArticleDOI
All-Inkjet-Printed 3D Alveolar Barrier Model with Physiologically Relevant Microarchitecture.
Dayoon Kang,Ju An Park,Woojo Kim,Seongju Kim,Hwa-Rim Lee,Woo-Jong Kim,Joo-Yeon Yoo,Sungjune Jung +7 more
TL;DR: In this paper, a 3D alveolar barrier model fabricated by printing four human alveocyte cell lines was presented, including type I and II cells, lung fibroblasts, lung microvascular endothelial cells, and HULEC-5a.
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The Interferon-Inducible Proteoglycan Testican-2/SPOCK2 Functions as a Protective Barrier against Virus Infection of Lung Epithelial Cells.
TL;DR: The data suggest that IFN-induced SPOCK2 functions as a decoy receptor to bind and block influenza virus infection, thereby restricting entry of the infecting virus into neighboring cells.