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Xiang Zeng

Researcher at Sun Yat-sen University

Publications -  54
Citations -  1582

Xiang Zeng is an academic researcher from Sun Yat-sen University. The author has contributed to research in topics: Spinal cord injury & Spinal cord. The author has an hindex of 21, co-authored 43 publications receiving 1078 citations. Previous affiliations of Xiang Zeng include Harvard University & VA Boston Healthcare System.

Papers
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Journal ArticleDOI

Bone marrow mesenchymal stem cells in a three-dimensional gelatin sponge scaffold attenuate inflammation, promote angiogenesis, and reduce cavity formation in experimental spinal cord injury.

Xiang Zeng, +7 more
- 01 Dec 2011 - 
TL;DR: In vivo transplantation experiments demonstrated that these scaffolds were biocompatible, and MSCs seeded to the scaffolds played an important role in attenuating inflammation, promoting angiogenesis, and reducing cavity formation.
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Targeted Nanotechnology in Glioblastoma Multiforme.

Talita Glaser, +3 more
- 31 Mar 2017 - 
TL;DR: A better understanding of CSC properties could result in the development of precise nanotherapies to fulfill unmet clinical needs, and novel biological strategies focused on manipulating gene expression for cancer therapy are analyzed.
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Fabrication and characterization of poly(l-lactic acid) 3D nanofibrous scaffolds with controlled architecture by liquid–liquid phase separation from a ternary polymer–solvent system

Liumin He, +8 more
- 31 Jul 2009 - 
TL;DR: In this paper, a 3D poly( l -lactic acid) scaffold with macro/micropores and nanofibrous structure was fabricated by phase separation from a ternary PLLA/dioxane/water system.
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Integration of donor mesenchymal stem cell-derived neuron-like cells into host neural network after rat spinal cord transection.

Xiang Zeng, +10 more
- 01 Jun 2015 - 
TL;DR: The data suggest that MSC-derived neuron-like cells resulting from NT-3-TrkC-induced differentiation can partially integrate into transected spinal cord and this strategy should be further investigated for reconstructing disrupted neural circuits.
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Pathophysiological Bases of Comorbidity: Traumatic Brain Injury and Post-Traumatic Stress Disorder.

Gary B. Kaplan, +14 more
- 03 Nov 2017 - 
TL;DR: The neural mechanisms underlying the TBI/PTSD comorbidity are analyzed and novel neurorestorative therapeutics using anti-inflammatory, antioxidant, and anticonvulsant agents may promote better outcomes forComorbid TBI and PTSD.