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Xing-Feng Yin

Researcher at Jinan University

Publications -  37
Citations -  1240

Xing-Feng Yin is an academic researcher from Jinan University. The author has contributed to research in topics: Phosphorylation & Proteomics. The author has an hindex of 17, co-authored 33 publications receiving 948 citations. Previous affiliations of Xing-Feng Yin include Sun Yat-sen University.

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Phosphoproteomic analysis reveals the multiple roles of phosphorylation in pathogenic bacterium Streptococcus pneumoniae.

TL;DR: A striking characteristic of S. pneumoniae phosphoproteome is the large number of multiple species-specific phosphorylated sites, indicating that high level of protein phosphorylation may play important roles in regulating many metabolic pathways and bacterial virulence.
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Translating mRNAs strongly correlate to proteins in a multivariate manner and their translation ratios are phenotype specific

TL;DR: These findings revealed, for the first time, the intrinsic and genome-wide translation modulations at translatomic level in human cells at steady-state, which are tightly correlated to the protein abundance and functionally relevant to cellular phenotypes.
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A hidden human proteome encoded by 'non-coding' genes.

TL;DR: A hidden human functional proteome encoded by purported lncRNAs is experimentally evidenced, suggesting a resource for annotating new human proteins.
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Global phosphoproteomic effects of natural tyrosine kinase inhibitor, genistein, on signaling pathways

TL;DR: Functional analysis suggested that genistein‐regulated protein tyrosine phosphorylation mainly by inhibiting the activity of tyrosin kinase EGFR, PDGFR, insulin receptor, Abl, Fgr, Itk, Fyn and Src can be functionally categorized into the canonial Receptor‐MAPK or Receptor-PI3K/AKT cascades.
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ACK1 promotes gastric cancer epithelial–mesenchymal transition and metastasis through AKT–POU2F1–ECD signalling

TL;DR: It is demonstrated that ACK1 promotes EMT, migration, and invasion by activating AKT–POU2F1–ECD signalling in GC cells and may be employed as a new prognostic factor and therapeutic target for GC.