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Xing-Feng Yin
Researcher at Jinan University
Publications - 37
Citations - 1240
Xing-Feng Yin is an academic researcher from Jinan University. The author has contributed to research in topics: Phosphorylation & Proteomics. The author has an hindex of 17, co-authored 33 publications receiving 948 citations. Previous affiliations of Xing-Feng Yin include Sun Yat-sen University.
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Phosphoproteomic analysis reveals the multiple roles of phosphorylation in pathogenic bacterium Streptococcus pneumoniae.
TL;DR: A striking characteristic of S. pneumoniae phosphoproteome is the large number of multiple species-specific phosphorylated sites, indicating that high level of protein phosphorylation may play important roles in regulating many metabolic pathways and bacterial virulence.
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Translating mRNAs strongly correlate to proteins in a multivariate manner and their translation ratios are phenotype specific
TL;DR: These findings revealed, for the first time, the intrinsic and genome-wide translation modulations at translatomic level in human cells at steady-state, which are tightly correlated to the protein abundance and functionally relevant to cellular phenotypes.
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A hidden human proteome encoded by 'non-coding' genes.
Shaohua Lu,Jing Zhang,Xinlei Lian,Xinlei Lian,Sun Li,Kun Meng,Yang Chen,Zhenghua Sun,Xing-Feng Yin,Yaxing Li,Jing Zhao,Tong Wang,Gong Zhang,Qing-Yu He +13 more
TL;DR: A hidden human functional proteome encoded by purported lncRNAs is experimentally evidenced, suggesting a resource for annotating new human proteins.
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Global phosphoproteomic effects of natural tyrosine kinase inhibitor, genistein, on signaling pathways
TL;DR: Functional analysis suggested that genistein‐regulated protein tyrosine phosphorylation mainly by inhibiting the activity of tyrosin kinase EGFR, PDGFR, insulin receptor, Abl, Fgr, Itk, Fyn and Src can be functionally categorized into the canonial Receptor‐MAPK or Receptor-PI3K/AKT cascades.
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ACK1 promotes gastric cancer epithelial–mesenchymal transition and metastasis through AKT–POU2F1–ECD signalling
Song-Hui Xu,Song-Hui Xu,Jin-Zhou Huang,Man-Li Xu,Guangchuang Yu,Xing-Feng Yin,De Chen,Guang-Rong Yan,Guang-Rong Yan +8 more
TL;DR: It is demonstrated that ACK1 promotes EMT, migration, and invasion by activating AKT–POU2F1–ECD signalling in GC cells and may be employed as a new prognostic factor and therapeutic target for GC.