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Yanqing Ding

Researcher at Southern Medical University

Publications -  157
Citations -  7721

Yanqing Ding is an academic researcher from Southern Medical University. The author has contributed to research in topics: Metastasis & Colorectal cancer. The author has an hindex of 40, co-authored 151 publications receiving 5870 citations.

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[A pathological report of three COVID-19 cases by minimal invasive autopsies].

TL;DR: The lungs from novel coronavirus pneumonia patients manifest significant pathological lesions, including theAlveolar exudative inflammation and interstitial inflammation, alveolar epithelium proliferation and hyaline membrane formation, while the 2019-nCoV is mainly distributed in lung, the infection also involves in the damages of heart, vessels, liver, kidney and other organs.
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Cancer-derived exosomal miR-25-3p promotes pre-metastatic niche formation by inducing vascular permeability and angiogenesis.

TL;DR: It is shown that colorectal cancer (CRC) derived exosomal miR-25-3p promotes vascular leakiness and angiogenesis, CRC metastasis, and is upregulated in CRC pateints with metastasis.
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CAFs secreted exosomes promote metastasis and chemotherapy resistance by enhancing cell stemness and epithelial-mesenchymal transition in colorectal cancer

TL;DR: Inhibiting exosomal miR-92a-3p provides an alternative modality for the prediction and treatment of metastasis and chemotherapy resistance in CRC, and promotes the stemness, epithelial-mesenchymal transition (EMT), metastasisand chemotherapy resistance of CRC cells.
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Emerging roles of circRNA_001569 targeting miR-145 in the proliferation and invasion of colorectal cancer.

TL;DR: It is shown that hsa_circ_001569 acted as a positive regulator in cell proliferation and invasion of colorectal cancer (CRC) and was identified as a sponge of miR-145 and up-regulatedMiRNAs functional targets E2F5, BAG4 and FMNL2.
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Expression of elevated levels of pro-inflammatory cytokines in SARS-CoV-infected ACE2+ cells in SARS patients: relation to the acute lung injury and pathogenesis of SARS.

TL;DR: It is suggested that cells infected by SARS‐CoV produce elevated levels of PICs which may cause immuno‐mediated damage to the lungs and other organs, resulting in ALI and, subsequently, multi‐organ dysfunction, and application of P IC antagonists may reduce the severity and mortality of SARS.