scispace - formally typeset
Y

Yong-Deok Jeon

Researcher at Chonbuk National University

Publications -  38
Citations -  736

Yong-Deok Jeon is an academic researcher from Chonbuk National University. The author has contributed to research in topics: Colitis & Ulcerative colitis. The author has an hindex of 14, co-authored 33 publications receiving 507 citations. Previous affiliations of Yong-Deok Jeon include Wonkwang University.

Papers
More filters
Journal ArticleDOI

Puerarin inhibits inflammation and oxidative stress in dextran sulfate sodium-induced colitis mice model.

TL;DR: It is suggested that puerarin has anti-inflammatory and anti-oxidative effects in the mouse model of colitis and significantly inhibited inflammation through the down-regulation of nuclear factor-κB and the secretion of pro-inflammatory mediators.
Journal ArticleDOI

Vanillic acid inhibits inflammatory mediators by suppressing NF-κB in lipopolysaccharide-stimulated mouse peritoneal macrophages.

TL;DR: The findings indicate that vanillic acid inhibits LPS-induced production of tumor necrosis factor (TNF)-α and interleukin (IL)-6 and suppressed the activation of nuclear factor-kappa B (NF-κB) and caspase-1, indicating the potential use of this molecule in the treatment of inflammatory diseases.
Journal ArticleDOI

Korean Red Ginseng improves atopic dermatitis-like skin lesions by suppressing expression of proinflammatory cytokines and chemokines in vivo and in vitro

TL;DR: Korean Red Ginseng water extract may be a useful therapeutic agent for the treatment of allergic inflammatory diseases such as AD-like dermatitis through blockage of the MAPK and NF-κB pathway.
Journal ArticleDOI

Allium hookeri root protects oxidative stress-induced inflammatory responses and β-cell damage in pancreas of streptozotocin-induced diabetic rats

TL;DR: Evidence is provided that AH protects the inflammatory responses by modulated NF-кBp65 signaling pathway through activation of HO-1 in the pancreas of STZ-induced diabetic rats, showing pancreato-protective effects against the development of inflammation in the diabetic rats.