Y
Yoshiki Nishizawa
Researcher at Osaka City University
Publications - 6
Citations - 2087
Yoshiki Nishizawa is an academic researcher from Osaka City University. The author has contributed to research in topics: Calcification & Hyperphosphatemia. The author has an hindex of 6, co-authored 6 publications receiving 2002 citations. Previous affiliations of Yoshiki Nishizawa include University of Washington.
Papers
More filters
Journal ArticleDOI
Phosphate regulation of vascular smooth muscle cell calcification.
Shuichi Jono,Marc D. McKee,Charles E. Murry,Atsushi Shioi,Yoshiki Nishizawa,Katsuhito Mori,Hirotoshi Morii,Cecilia M. Giachelli +7 more
TL;DR: It is suggested that elevated phosphate may directly stimulate HSMCs to undergo phenotypic changes that predispose to calcification and offer a novel explanation of the phenomenon of vascular calcification under hyperphosphatemic conditions.
Journal ArticleDOI
Vascular calcification and inorganic phosphate
Cecilia M. Giachelli,Shuichi Jono,Atshushi Shioi,Yoshiki Nishizawa,Katshuhito Mori,Hirotoshi Morii +5 more
TL;DR: Elevated Pi levels may directly stimulate HSMC to undergo phenotypic changes that predispose to calcification and may help explain both the phenomena of human metastatic calcification under hyperphosphatemic conditions as well as increased calcification in PDGF-rich atherosclerotic lesions.
Journal ArticleDOI
FGF-23 in patients with end-stage renal disease on hemodialysis
Yasuo Imanishi,Masaaki Inaba,Kiyoshi Nakatsuka,Kyoko Nagasue,Senji Okuno,Asami Yoshihara,Masakazu Miura,Akimitsu Miyauchi,Keisuke Kobayashi,Takami Miki,Tetsuo Shoji,Eiji Ishimura,Yoshiki Nishizawa +12 more
TL;DR: Serum phosphate, calcium, and intact PTH could be regulators of FGF-23 levels in uremic patients on maintenance hemodialysis and may provide new insights into the pathophysiologic effects of F GF-23 on calcium-phosphate homeostasis.
Journal ArticleDOI
Vascular calcification in chronic kidney disease.
TL;DR: It is suggested that the balance of inducers, such as phosphate, and inhibitors,such as MGP, fetuin-A, and others, are likely to control whether or not calcification occurs under pathological conditions, and that bone-associated proteins may be involved in the development of vascular calcification.
Journal ArticleDOI
Mönckeberg's medial sclerosis and inorganic phosphate in uremia
Atsushi Shioi,Hiromichi Taniwaki,Shuichi Jono,Yasuhisa Okuno,Hidenori Koyama,Katsuhito Mori,Yoshiki Nishizawa +6 more
TL;DR: Hyperphosphatemia among various metabolic disturbances recognized in Uremia may play an important role in the development of MMS in uremia.