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Yukitaka Shizukuda
Researcher at University of Cincinnati
Publications - 69
Citations - 3201
Yukitaka Shizukuda is an academic researcher from University of Cincinnati. The author has contributed to research in topics: Hereditary hemochromatosis & Asymptomatic. The author has an hindex of 22, co-authored 66 publications receiving 3018 citations. Previous affiliations of Yukitaka Shizukuda include University of Illinois at Chicago & University of Cincinnati Academic Health Center.
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Journal ArticleDOI
Pulmonary hypertension as a risk factor for death in patients with sickle cell disease
Mark T. Gladwin,Vandana Sachdev,Maria Jison,Yukitaka Shizukuda,Jonathan F. Plehn,Karin R. Minter,Bernice Brown,Wynona Coles,James S. Nichols,Inez Ernst,Lori A. Hunter,William C. Blackwelder,Alan N. Schechter,Griffin P. Rodgers,Oswaldo Castro,Frederick P. Ognibene +15 more
TL;DR: Pulmonary hypertension appears to be a complication of chronic hemolysis, is resistant to hydroxyurea therapy, and confers a high risk of death in adults with sickle cell disease.
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Diastolic dysfunction is an independent risk factor for death in patients with sickle cell disease.
Vandana Sachdev,Roberto F. Machado,Yukitaka Shizukuda,Yesoda N. Rao,Stanislav Sidenko,Inez Ernst,Marilyn St. Peter,Wynona Coles,Douglas R. Rosing,William C. Blackwelder,Oswaldo Castro,Gregory J. Kato,Mark T. Gladwin +12 more
TL;DR: Data from this study support the implementation of echocardiographic screening of adult patients with SCD to identify high-risk individuals for further evaluation and contribute independently to prospective mortality in patients withSCD.
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Iron Overload Cardiomyopathy, Better Understanding of An Increasing Disorder
TL;DR: There is a need for the development of clinical guidelines in order to improve the management of this emerging complex disease.
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Vascular Endothelial Growth Factor–Induced Endothelial Cell Migration and Proliferation Depend on a Nitric Oxide–Mediated Decrease in Protein Kinase Cδ Activity
TL;DR: In this article, the importance of the activity of specific protein kinase C (PKC) isoenzymes in human EC migration and proliferation in response to VEGF was tested.
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β-Adrenergic stimulation causes cardiocyte apoptosis: Influence of tachycardia and hypertrophy
Yukitaka Shizukuda,Peter M. Buttrick,Peter M. Buttrick,David L. Geenen,Alain C. Borczuk,Richard N. Kitsis,Edmund H. Sonnenblick +6 more
TL;DR: Catecholamines, in the absence of altered systolic load, induce Apo which is not mediated solely by tachycardia, but left ventricular hypertrophy secondary to abdominal aortic banding is associated with Apo, but this does not increase sensitivity to isoproterenol-induced Apo.