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Yves Lecarpentier

Researcher at University of Paris

Publications -  90
Citations -  2641

Yves Lecarpentier is an academic researcher from University of Paris. The author has contributed to research in topics: Blood pressure & Isometric exercise. The author has an hindex of 23, co-authored 90 publications receiving 2539 citations. Previous affiliations of Yves Lecarpentier include University of Paris-Sud & École Polytechnique.

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Journal ArticleDOI

New formula for predicting mean pulmonary artery pressure using systolic pulmonary artery pressure.

Denis Chemla, +6 more
- 01 Oct 2004 - 
TL;DR: In resting humans, MPAP can be accurately predicted from SPAP over a wide pressure range, and a new formula proposed may help to refine the threshold pressure values used in the diagnosis of PH.
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Myosin isoenzymic distribution correlates with speed of myocardial contraction.

Ketty Schwartz, +7 more
- 01 Dec 1981 - 
Journal ArticleDOI

Haemodynamic evaluation of pulmonary hypertension

Denis Chemla, +4 more
- 01 Nov 2002 - 
TL;DR: Pulmonary hypertension is characterised by the chronic elevation of pulmonary artery pressure (PAP) and pulmonary vascular resistance (PVR) leading to right ventricular enlargement and hypertrophy, which is associated with poor prognosis and right heart failure.
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Pulmonary artery pulse pressure and wave reflection in chronic pulmonary thromboembolism and primary pulmonary hypertension

Vincent Castelain, +5 more
- 15 Mar 2001 - 
TL;DR: CPTE and PPH with severe pulmonary hypertension had similar PA pulse pressure, and wave reflection is elevated in both groups, and CPTE had increased and anticipated wave reflection as compared with PPH, thus suggesting differences in the pulsatile component of right ventricular afterload.
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Effects of chronic growth hormone hypersecretion on intrinsic contractility, energetics, isomyosin pattern, and myosin adenosine triphosphatase activity of rat left ventricle.

José Timsit, +9 more
- 01 Aug 1990 - 
TL;DR: It is demonstrated that chronic GH hypersecretion in the rat leads to a unique pattern of myocardial adaptation which allows the muscle to improve its contractile performance and economy simultaneously, thanks to myosin phenoconversion and an increase in the number of active enzymatic sites.