Z
Zannatul Ferdous
Researcher at United Arab Emirates University
Publications - 5
Citations - 540
Zannatul Ferdous is an academic researcher from United Arab Emirates University. The author has contributed to research in topics: Voltage-gated potassium channel & Voltage-dependent calcium channel. The author has an hindex of 4, co-authored 5 publications receiving 192 citations.
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Health Impact of Silver Nanoparticles: A Review of the Biodistribution and Toxicity Following Various Routes of Exposure
TL;DR: The present review discusses the current advances on the physiochemical properties of AgNPs with specific emphasis on biodistribution and both in vitro and in vivo toxicity following various routes of exposure.
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The in Vitro Effect of Polyvinylpyrrolidone and Citrate Coated Silver Nanoparticles on Erythrocytic Oxidative Damage and Eryptosis
TL;DR: Both PVP- and CT- AgNPs causes hemolysis, and are taken up by erythrocytes, and these findings provide evidence for the potential pathophysiological effect of PVP -and CT-AgNPs on ERYthrocyte physiology.
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Pulmonary exposure to silver nanoparticles impairs cardiovascular homeostasis: Effects of coating, dose and time.
TL;DR: Pulmonary exposure to silver nanoparticles caused cardiac oxidative stress, DNA damage and apoptosis, alteration of coagulation markers and thrombosis, and a novel mechanistic insight into the cardiovascular pathophysiological effects of lung exposure to AgNPs are investigated.
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Different Profile of mRNA Expression in Sinoatrial Node from Streptozotocin-Induced Diabetic Rat.
Zannatul Ferdous,Muhammad Anwar Qureshi,Petrilla Jayaprakash,K. Parekh,Annie John,Murat Oz,Haider Raza,Halina Dobrzynski,Thomas E. Adrian,Frank Christopher Howarth +9 more
TL;DR: Differences in the profile of mRNA encoding a variety of proteins that are associated with the generation, conduction and regulation of electrical signals in the SAN of STZ-induced diabetic rat heart are demonstrated.
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Remote effects and biodistribution of pulmonary instilled silver nanoparticles in mice
TL;DR: Pulmonary exposure to AgNPs caused oxidative stress markers, inflammation, DNA damage and biodistribution in remote organs, and these findings provide a novel mechanistic insight into the pathophysiological effects and tissue distribution of lung exposure toAgNPs.