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Zoran Marković

Researcher at University of Belgrade

Publications -  192
Citations -  5147

Zoran Marković is an academic researcher from University of Belgrade. The author has contributed to research in topics: Fullerene & Graphene. The author has an hindex of 29, co-authored 177 publications receiving 4173 citations. Previous affiliations of Zoran Marković include Slovak Academy of Sciences & American Museum of Natural History.

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In vitro comparison of the photothermal anticancer activity of graphene nanoparticles and carbon nanotubes.

TL;DR: Graphene nanoparticles performed significantly better than CNT in inducing photothermal death of U251 human glioma cells in vitro and could be largely explained by their better dispersivity, which has been supported by a simple calculation.
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Biomedical potential of the reactive oxygen species generation and quenching by fullerenes (C60)

TL;DR: The mechanisms and biological consequences of ROS generation/quenching by C60 are analyzed, focusing on the influence that different physico-chemical alterations exert on its ROS-related biological behavior.
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Graphene quantum dots as autophagy-inducing photodynamic agents

TL;DR: Electrochemically produced GQD irradiated with blue light generates reactive oxygen species, including singlet oxygen, and kill U251 human glioma cells by causing oxidative stress and a genetic inactivation of autophagy-essential LC3B protein partly abrogated the photodynamic cytotoxicity of G QD.
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Photodynamic antibacterial effect of graphene quantum dots.

TL;DR: Electrochemically produced graphene quantum dots (GQD), a new class of carbon nanoparticles, generate reactive oxygen species when photoexcited, and kill two strains of pathogenic bacteria, methicillin-resistant Staphylococcus aureus and Escherichia coli.
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Distinct cytotoxic mechanisms of pristine versus hydroxylated fullerene.

TL;DR: Unlike polyhydroxylated C60 that exerts mainly antioxidant/cytoprotective and only mild ROS-independent pro-apoptotic activity, pure crystalline C60 seems to be endowed with strong pro-oxidant capacity responsible for the rapid necrotic cell death.