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Evaluation of Carotenoid Concentration in Chicken Tissues

Howard A. Stone, +2 more
- 01 May 1971 - 
- Vol. 50, Iss: 3, pp 675-681
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TLDR
Genetic studies involving yellow-skinned chickens only, have indicated that genotype and sex influence the subjective score in pigmentation, and xanthophyll and lutein were the principal natural yellow pigments of the chicken skin and shank.
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This article is published in Poultry Science.The article was published on 1971-05-01 and is currently open access. It has received 18 citations till now. The article focuses on the topics: Lutein.

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Parasites and mate choice in red jungle fowl.

TL;DR: Captive flocks of red jungle fowl experimentally infected with the intestinal nematode Ascaridia galli were used to test Hamilton and Zuk's hypothesis that parasites adversely affect male secondary sex characters and that females prefer unparasitized over parasitized males and that parasites have a disproportionately larger effect on ornamental traits.
Journal ArticleDOI

Better red than dead: carotenoid-based mouth coloration reveals infection in barn swallow nestlings.

TL;DR: It is demonstrated that the gape colour of nestlings challenged with a novel antigen and provided with the principal circulating carotenoid (lutein) have more brightly coloured red gapes than their challenged but unsupplemented siblings and this suggests that parents may favour nestlings with superior health by preferentially feeding offspring with the brightest gapes.
Journal ArticleDOI

Intraspecific variation in plumage coloration reflects immune-response in great tit (parus-major) males

R. Dufva, +1 more
- 01 Oct 1995 - 
TL;DR: The view that brighter males signal low parasitation intensity as a result of their superior immunity is favoured, as it is well known that the number and proportions of different types of leucocytes reflect the health status of individuals, as these cells quickly respond to stress and infectious diseases.
References
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Genetics of the fowl

TL;DR: Partial suppression of the frizzled plumage of heterozygous Frizzles is induced by a recessive autosomal gene, mf, which is independent of the gene for frizzling as discussed by the authors.
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