Open AccessJournal Article
Oxygen poisoning in mice. Ultrastructural and surfactant studies during exposure and recovery.
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This article is published in Archives of pathology.The article was published on 1970-11-01 and is currently open access. It has received 82 citations till now. The article focuses on the topics: Pulmonary surfactant & Histocytochemistry.read more
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Journal ArticleDOI
Sequential changes in lung morphology during the repair of acute oxygen-induced lung injury in adult rats.
TL;DR: The repair of oxygen-induced lung injury involves a complex pattern of morphologic changes that has important similarities to those occurring during repair on other tissues such as the skin.
Journal ArticleDOI
Effects of 4-ipomeanol, a product from mold-damaged sweet potatoes, on the bovine lung.
TL;DR: Cattle given intraruminal administration of 4-ipomeanol, a furanoterpenoid originally obtained from sweet potatoes infected with Fusarium solani, developed a respiratory syndrome clinically and histologically indistinguishable from atypical interstitial pneumonia.
Journal ArticleDOI
Genetic basis of murine responses to hyperoxia-induced lung injury
Gregory S. Whitehead,Lauranell H. Burch,Katherine G. Berman,Claude A. Piantadosi,David A. Schwartz,David A. Schwartz,David A. Schwartz +6 more
TL;DR: The data suggest that the response to O2 depends, in part, on the genetic background and that some of the strains analyzed can be used to identify specific loci and genes underlying the responseto O2.
Journal ArticleDOI
Contribution of proliferation and DNA damage repair to alveolar epithelial type 2 cell recovery from hyperoxia
TL;DR: It is demonstrated that in healthy, adult male C57BL/6J mice, AEC2 proliferation, damage, and repair follow separate courses during hyperoxia recovery and that both proliferation and efficient repair may be required to ensure A EC2 survival.
Journal ArticleDOI
The effect of oxygen tension on rat hepatocytes in short-term culture
TL;DR: It was additionally demonstrated that lipid peroxidation was consistantly lower in hepatocytes cultured in air:N2 as compared to air or air:O2, which may be related to oxidative cell damage.
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