Journal ArticleDOI
The treatment of burn shock by the intravenous and oral administration of hypertonic lactated saline solution.
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This article is published in Journal of Trauma-injury Infection and Critical Care.The article was published on 1970-07-01. It has received 129 citations till now. The article focuses on the topics: Saline & Oral administration.read more
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Journal ArticleDOI
American Burn Association practice guidelines burn shock resuscitation.
TL;DR: There are insufficient data to support a treatment standard treatment at this time, and the addition of colloid-containing fluid following burn injury, especially after the first 12 to 24 hours postburn, may decrease overall fluid requirements.
Journal ArticleDOI
The phenomenon of fluid creep in acute burn resuscitation
TL;DR: Strategies to reduce fluid creep include the avoidance of early overresuscitation, use of colloid as a routine component of resuscitation or for “rescue,” and adherence to protocols for fluid resuscitation.
Journal ArticleDOI
Current treatment of severely burned patients.
TL;DR: A review of studies and clinical trials from the past to the present include fluid resuscitation, sepsis, immune function, hypermetabolism, early excision, wound healing, scar formation, and inhalation injury.
Journal ArticleDOI
Hypertonic sodium resuscitation is associated with renal failure and death.
TL;DR: Hypertonic sodium solution resuscitation of burn patients did not reduce the total resuscitation volume required and was associated with an increased incidence of renal failure and death.
Journal ArticleDOI
Hypertonic lactated saline resuscitation reduces the risk of abdominal compartment syndrome in severely burned patients.
Jun Oda,Masashi Ueyama,Katsuyuki Yamashita,Takuya Inoue,Mitsuhiro Noborio,Yasumasa Ode,Yoshiki Aoki,Hisashi Sugimoto +7 more
TL;DR: The data suggest that HLS resuscitation could reduce the risk of secondary abdominal compartment syndrome with lower fluid load in burn shock patients, and a large intravenous fluid volume decreases abdominal perfusion during the resuscitative period because of increased IAP.
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Physiological response to crystalloid resuscitation of severe burns.
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