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A. Jay Gandolfi
Researcher at University of Arizona
Publications - 141
Citations - 5223
A. Jay Gandolfi is an academic researcher from University of Arizona. The author has contributed to research in topics: Halothane & Arsenite. The author has an hindex of 41, co-authored 141 publications receiving 4876 citations. Previous affiliations of A. Jay Gandolfi include United States Geological Survey.
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Journal ArticleDOI
Rice consumption contributes to arsenic exposure in US women
Diane Gilbert-Diamond,Kathryn L. Cottingham,Joann F. Gruber,Tracy Punshon,Vicki Sayarath,A. Jay Gandolfi,Emily R Baker,Brian P. Jackson,Carol L. Folt,Margaret R. Karagas +9 more
TL;DR: A positive association between rice consumption and urinary arsenic excretion, a biomarker of recent arsenic exposure, in 229 pregnant women is documented, indicating that rice consumption should be considered when designing arsenic reduction strategies in the United States.
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Precision-cut tissue slices: applications in pharmacology and toxicology.
TL;DR: Significant advances in the methods to produce and culture tissue slices have been made, as well as the application of the technique to several other organs, including kidney, lung and heart, are made.
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Anaerobic biotransformation of roxarsone and related N-substituted phenylarsonic acids
Irail Cortinas,Jim A. Field,Mike Kopplin,John R. Garbarino,A. Jay Gandolfi,Reyes Sierra-Alvarez +5 more
TL;DR: It is demonstrated that roxarsone is rapidly transformed in the absence of oxygen to the corresponding aromatic amine, 4-hydroxy-3-aminophenylarsonic acid (HAPA), the first report of a biologically catalyzed rupture of the phenylarsic group under anaerobic conditions.
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The metabolism of inorganic arsenic oxides, gallium arsenide, and arsine: a toxicochemical review
TL;DR: There is insufficient evidence to equate the different arsenic compounds, and there are several differences in the toxicity of the arsenic compounds that will require substantial research.
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Epigenetic remodeling during arsenical-induced malignant transformation
TL;DR: Changes in histone H3 acetylation occur during arsenical-induced malignant transformation that are linked to the expression state of the associated gene and suggest that arsenicals may participate in tumorigenesis by altering the epigenetic terrain of select genes.