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Abdulaziz M. Aleisa
Researcher at King Saud University
Publications - 62
Citations - 3174
Abdulaziz M. Aleisa is an academic researcher from King Saud University. The author has contributed to research in topics: Long-term potentiation & Nicotine. The author has an hindex of 34, co-authored 62 publications receiving 2876 citations. Previous affiliations of Abdulaziz M. Aleisa include University of Houston.
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Journal ArticleDOI
Design, synthesis and biological evaluation of novel quinazoline derivatives as potential antitumor agents: Molecular docking study
Adel S. El-Azab,Mohamed A. Al-Omar,Alaa A.-M. Abdel-Aziz,Naglaa I. Abdel-Aziz,Magda A.-A. El-Sayed,Abdulaziz M. Aleisa,Mohamed M. Sayed-Ahmed,Sami G. Abdel-Hamide +7 more
TL;DR: Novel derivatives of quinazoline have been synthesized and tested for their antitumor activity against three tumor cell lines among these cell lines the human breast carcinoma cell line (MCF-7) in which EGFR is highly expressed.
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Thymoquinone attenuates diethylnitrosamine induction of hepatic carcinogenesis through antioxidant signaling
Mohamed M. Sayed-Ahmed,Abdulaziz M. Aleisa,Salim S. Al-Rejaie,Abdulaziz A. Al-Yahya,Othman A. Al-Shabanah,Mohamed M. Hafez,Mahmoud N. Nagi +6 more
TL;DR: TQ supplementation prevents the development of DENA-induced initiation of liver cancer by decreasing oxidative stress and preserving both the activity and mRNA expression of antioxidant enzymes.
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Chronic caffeine treatment prevents sleep deprivation-induced impairment of cognitive function and synaptic plasticity.
TL;DR: The results suggest that long-term use of a low dose of caffeine prevents impairment of short-term memory and E-LTP in acutely sleep-deprived rats.
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Levothyroxin restores hypothyroidism-induced impairment of hippocampus-dependent learning and memory: Behavioral, electrophysiological, and molecular studies.
TL;DR: Thyroxin treatment reverses hypothyroidism‐induced impairment of hippocampus‐dependent cognition, and L‐LTP, probably by restoring the levels of signaling molecule important for these processes.
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Sleep deprivation prevents stimulation-induced increases of levels of P-CREB and BDNF: protection by caffeine.
TL;DR: The results suggest that chronic caffeine treatment may protect the sleep-deprived brain probably by preserving the levels of P-CREB and BDNF.