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Adam Deak

Researcher at University of Debrecen

Publications -  40
Citations -  257

Adam Deak is an academic researcher from University of Debrecen. The author has contributed to research in topics: Hemorheology & Erythrocyte deformability. The author has an hindex of 8, co-authored 35 publications receiving 190 citations. Previous affiliations of Adam Deak include Rutgers University.

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A2A adenosine receptors control pancreatic dysfunction in high-fat-diet-induced obesity

TL;DR: Ex vivo islet experiments confirmed the role of A2A ARs in protecting against decreased insulin content and release caused by HFD, and experiments with bone marrow chimeras revealed that inflammation was not the primary cause of decreased insulin secretion in A 2A AR‐KO mice.
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Hypothalamic S-nitrosylation contributes to the counter-regulatory response impairment following recurrent hypoglycemia.

TL;DR: Pretreatment with the anti-oxidant N-acetyl-cysteine (NAC) was used to prevent increased VMH S-nitrosylation and the data suggest that NAC may be clinically useful in preventing impaired CRR in patients undergoing intensive-insulin therapy.
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Effects and influencing factors on hemorheological variables taken into consideration in surgical pathophysiology research.

TL;DR: This paper aimed to make an attempt on summarizing the possible factors and conditions that might have an effect on hemorheological results in experimental surgical studies.
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Effect of unilateral labyrinthectomy on the molecular composition of perineuronal nets in the lateral vestibular nucleus of the rat

TL;DR: It is demonstrated for the first time that the UL is accompanied by the modification of the HA, and CSPG staining pattern in the PNN of the LVN in the rat following UL, implying the role of HA and C SPGs in the vestibular compensation.
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Early hemorheological changes in a porcine model of intravenously given E. coli induced fulminant sepsis.

TL;DR: Over the experimental period, the tendency of impairment in erythrocyte deformability and the controversial decreasing of ery Throcyte aggregation values were well detected in this porcine model during the early hours of fulminant sepsis.