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Alberto Monescillo

Researcher at Hospital Universitario Insular de Gran Canaria

Publications -  13
Citations -  2915

Alberto Monescillo is an academic researcher from Hospital Universitario Insular de Gran Canaria. The author has contributed to research in topics: Cirrhosis & Ascites. The author has an hindex of 10, co-authored 13 publications receiving 2666 citations. Previous affiliations of Alberto Monescillo include University of Alcalá.

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Randomized trial comparing albumin, dextran 70, and polygeline in cirrhotic patients with ascites treated by paracentesis

TL;DR: Postparacentesis circulatory dysfunction is not spontaneously reversible and is associated with a shorter time to first readmission and shorter survival, and albumin is the best plasma expander to prevent this complication.
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Circulatory function and hepatorenal syndrome in cirrhosis.

TL;DR: The data indicate that hepatorenal syndrome is the result of a decrease in cardiac output in the setting of a severe arterial vasodilation, and plasma renin activity and cardiac output were the only independent predictors of hepat Lorenal syndrome.
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Terlipressin and albumin vs albumin in patients with cirrhosis and hepatorenal syndrome: a randomized study.

TL;DR: Treatment with terlipressin and albumin is effective in improving renal function in patients with cirrhosis and hepatorenal syndrome and survival at 3 months was not significantly different between the groups.
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Influence of portal hypertension and its early decompression by TIPS placement on the outcome of variceal bleeding

TL;DR: It is concluded that increased portal pressure estimated by early HVPG measurement is a main determinant of treatment failure and survival in variceal bleeding, and early TIPS placement reduces treatment failures and mortality in high risk patients defined by hemodynamic criteria.
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Systemic, renal, and hepatic hemodynamic derangement in cirrhotic patients with spontaneous bacterial peritonitis

TL;DR: Patients with SBP frequently develop a rapidly progressive impairment in systemic hemodynamics, leading to severe renal and hepatic failure, aggravation of portal hypertension, encephalopathy, and death, despite rapid resolution of infection.